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pubmed-article:9431893pubmed:abstractTextThe major experimental and intellectual advance in gastroenterology in recent years has been the observation that mice with deletions in specific cytokine genes develop a spontaneous inflammatory bowel disease. Inflammatory bowel disease has also been seen in a number of other mouse strains with perturbed immune responses. The driving force for disease in a number of these models is the normal bacterial flora. These studies show that the gut immune system is in homeostasis with the antigens of the flora, but inappropriate T-helper 1 (Th1)-type responses to the flora cause severe gut injury and since the antigenic stimulus persists, the consequence is chronic inflammation.lld:pubmed
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pubmed-article:9431893pubmed:articleTitleCytokine gene deleted mice in the study of gastrointestinal inflammation.lld:pubmed
pubmed-article:9431893pubmed:affiliationDepartment of Paediatric Gastroenterology, St Bartholomew's and the Royal London School of Medicine and Dentistry, St Bartholomew's Hospital, UK.lld:pubmed
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