pubmed-article:9352872 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9352872 | lifeskim:mentions | umls-concept:C0220847 | lld:lifeskim |
pubmed-article:9352872 | lifeskim:mentions | umls-concept:C0042769 | lld:lifeskim |
pubmed-article:9352872 | lifeskim:mentions | umls-concept:C0019630 | lld:lifeskim |
pubmed-article:9352872 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:9352872 | lifeskim:mentions | umls-concept:C1274040 | lld:lifeskim |
pubmed-article:9352872 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:9352872 | pubmed:dateCreated | 1997-11-13 | lld:pubmed |
pubmed-article:9352872 | pubmed:abstractText | The host's immune response may influence the course of hepatitis C virus (HCV) infection. The aim of this study was to investigate the distribution of HLA class II alleles in white subjects who spontaneously recovered from HCV infection compared with that in patients with persistent infection. | lld:pubmed |
pubmed-article:9352872 | pubmed:language | eng | lld:pubmed |
pubmed-article:9352872 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9352872 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:9352872 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9352872 | pubmed:month | Nov | lld:pubmed |
pubmed-article:9352872 | pubmed:issn | 0016-5085 | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:AbbalMM | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:DuffautMM | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:PuelJJ | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:PascalJ PJP | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:CharletJ PJP | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:VinelJ PJP | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:SelvesJJ | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:IzopetJJ | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:FortMM | lld:pubmed |
pubmed-article:9352872 | pubmed:author | pubmed-author:AlricLL | lld:pubmed |
pubmed-article:9352872 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9352872 | pubmed:volume | 113 | lld:pubmed |
pubmed-article:9352872 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9352872 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9352872 | pubmed:pagination | 1675-81 | lld:pubmed |
pubmed-article:9352872 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:9352872 | pubmed:year | 1997 | lld:pubmed |
pubmed-article:9352872 | pubmed:articleTitle | Genes of the major histocompatibility complex class II influence the outcome of hepatitis C virus infection. | lld:pubmed |
pubmed-article:9352872 | pubmed:affiliation | Service de Médecine Interne, Hôpital Purpan, INSERM Unité CJF-9107, Toulouse, France. | lld:pubmed |
pubmed-article:9352872 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9352872 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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