| pubmed-article:8536990 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C0178453 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C0025186 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C0598501 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C1424613 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C1523479 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C0597767 | lld:lifeskim |
| pubmed-article:8536990 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:8536990 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8536990 | pubmed:dateCreated | 1996-2-8 | lld:pubmed |
| pubmed-article:8536990 | pubmed:abstractText | The fission yeast Schizosaccharomyces pombe does not form tripartite synaptonemal complexes during meiotic prophase, but axial core-like structures (linear elements). To probe the relationship between meiotic recombination and the structure, pairing, and segregation of meiotic chromosomes, we genetically and cytologically characterized the rec8-110 mutant, which is partially deficient in meiotic recombination. The pattern of spore viability indicates that chromosome segregation is affected in the mutant. A detailed segregational analysis in the rec8-110 mutant revealed more spores disomic for chromosome III than in a wild-type strain. Aberrant segregations are caused by precocious segregation of sister chromatids at meiosis I, rather than by nondisjunction as a consequence of lack of crossovers. In situ hybridization further showed that the sister chromatids are separated prematurely during meiotic prophase. Moreover, the mutant forms aberrant linear elements and shows a shortened meiotic prophase. Meiotic chromosome pairing in interstitial and centromeric regions is strongly impaired in rec8-110, whereas the chromosome ends are less deficient in pairing. We propose that the rec8 gene encodes a protein required for linear element formation and that the different phenotypes of rec8-110 reflect direct and indirect consequences of the absence of regular linear elements. | lld:pubmed |
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| pubmed-article:8536990 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8536990 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8536990 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8536990 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8536990 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8536990 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:8536990 | pubmed:author | pubmed-author:MolnarMM | lld:pubmed |
| pubmed-article:8536990 | pubmed:author | pubmed-author:KohliJJ | lld:pubmed |
| pubmed-article:8536990 | pubmed:author | pubmed-author:SipiczkiMM | lld:pubmed |
| pubmed-article:8536990 | pubmed:author | pubmed-author:BählerJJ | lld:pubmed |
| pubmed-article:8536990 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8536990 | pubmed:volume | 141 | lld:pubmed |
| pubmed-article:8536990 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8536990 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8536990 | pubmed:pagination | 61-73 | lld:pubmed |
| pubmed-article:8536990 | pubmed:dateRevised | 2010-9-10 | lld:pubmed |
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| pubmed-article:8536990 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:8536990 | pubmed:articleTitle | The rec8 gene of Schizosaccharomyces pombe is involved in linear element formation, chromosome pairing and sister-chromatid cohesion during meiosis. | lld:pubmed |
| pubmed-article:8536990 | pubmed:affiliation | Department of Genetics, University of Debrecen, Hungary. | lld:pubmed |
| pubmed-article:8536990 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8536990 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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