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pubmed-article:8422586pubmed:abstractTextWe report the following observations: first that cell body damage induced by ibotenic acid within the medial region of the amygdala resulted in the abolition of corticosterone-potentiated, aldosterone-induced salt appetite. Sodium depletion-induced salt appetite was not impaired by the lesion, nor was angiotensin-dependent salt appetite via captopril treatment. These results provide evidence that the medial region of the amygdala has a major role in the natriorexigenic effects of the adrenal steroids.lld:pubmed
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pubmed-article:8422586pubmed:articleTitleMedial region of the amygdala: involvement in adrenal-steroid-induced salt appetite.lld:pubmed
pubmed-article:8422586pubmed:affiliationDepartment of Biology, University of Pennsylvania, Philadelphia.lld:pubmed
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