| pubmed-article:8106101 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8106101 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:8106101 | lifeskim:mentions | umls-concept:C0007776 | lld:lifeskim |
| pubmed-article:8106101 | lifeskim:mentions | umls-concept:C0013030 | lld:lifeskim |
| pubmed-article:8106101 | lifeskim:mentions | umls-concept:C0206128 | lld:lifeskim |
| pubmed-article:8106101 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8106101 | pubmed:dateCreated | 1993-12-14 | lld:pubmed |
| pubmed-article:8106101 | pubmed:abstractText | Slices from fresh specimens of human neocortex which had to be removed during neurosurgery to reach subcortical tumours were labelled with [3H]-dopamine and stimulated electrically. Quinpirole, a selective dopamine D2 receptor agonist, inhibited the stimulated tritium overflow (EC50 = 25 nM; maximal inhibition: about 80% at 10 microM). The selective D1 receptor agonist, SKF 38393, was inactive up to 10 microM. Quinpirole was antagonized by the D2 receptor antagonist (-)-sulpiride (apparent pA2 = 8.26). Thus dopaminergic axon terminals in the human mesocortical pathway possess autoreceptors of the D2 type. | lld:pubmed |
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| pubmed-article:8106101 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8106101 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8106101 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8106101 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:8106101 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8106101 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8106101 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8106101 | pubmed:issn | 0007-1188 | lld:pubmed |
| pubmed-article:8106101 | pubmed:author | pubmed-author:RaiteriMM | lld:pubmed |
| pubmed-article:8106101 | pubmed:author | pubmed-author:AndrioliG CGC | lld:pubmed |
| pubmed-article:8106101 | pubmed:author | pubmed-author:FedeleEE | lld:pubmed |
| pubmed-article:8106101 | pubmed:author | pubmed-author:RuelleAA | lld:pubmed |
| pubmed-article:8106101 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8106101 | pubmed:volume | 110 | lld:pubmed |
| pubmed-article:8106101 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8106101 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8106101 | pubmed:pagination | 20-2 | lld:pubmed |
| pubmed-article:8106101 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:8106101 | pubmed:meshHeading | pubmed-meshheading:8106101-... | lld:pubmed |
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| pubmed-article:8106101 | pubmed:meshHeading | pubmed-meshheading:8106101-... | lld:pubmed |
| pubmed-article:8106101 | pubmed:year | 1993 | lld:pubmed |
| pubmed-article:8106101 | pubmed:articleTitle | Release-regulating dopamine autoreceptors in human cerebral cortex. | lld:pubmed |
| pubmed-article:8106101 | pubmed:affiliation | Institute of Pharmacology and Pharmacognosy, University of Genova, Italy. | lld:pubmed |
| pubmed-article:8106101 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8106101 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:8106101 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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