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pubmed-article:7895352pubmed:abstractTextAlthough extensive evidence indicates that estrogen is responsible for the markedly decreased cardiovascular risk of premenopausal women, the mechanism through which estrogen might exert its protective effect has not been adequately explained. Since thrombosis is now recognized to play an important role in the onset of cardiovascular disease, we investigated the relation between estrogen status and fibrinolytic potential, a determinant of thrombotic risk.lld:pubmed
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pubmed-article:7895352pubmed:articleTitleAssociation between increased estrogen status and increased fibrinolytic potential in the Framingham Offspring Study.lld:pubmed
pubmed-article:7895352pubmed:affiliationInstitute for Prevention of Cardiovascular Disease, Deaconess Hospital, Boston, Mass 02215.lld:pubmed
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