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pubmed-article:7865481pubmed:abstractTextPrevious studies from our laboratories have shown that UV-B irradiation at the site of an intradermal infection of herpes simplex virus (HSV) resulted in a higher incidence of zosteriform lesions and suppressed cellular immune responses to HSV in mice. In order to determine whether the production of T-cell-derived cytokine (IFN-gamma, IL-2 and IL-4) by immune cells from irradiated mice is also suppressed, we examined the production of cytokines by lymph node cells and spleen cells taken from UV-B irradiated, HSV type 1 (HSV-1)-infected mice. UV-B irradiation (120 mJ/cm2) prior to HSV-1 infection was found to markedly suppress IFN-gamma production compared to that of the non-irradiated control. IL-4 production was enhanced compared to IL-2 in the UV-B irradiated mice. These results suggest that alteration(s) in the cytokine production profile may therefore be involved in the development of severe skin lesions caused by HSV infection in UV-B irradiated mice.lld:pubmed
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pubmed-article:7865481pubmed:dateRevised2008-11-21lld:pubmed
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pubmed-article:7865481pubmed:articleTitleUltraviolet-B irradiation alters cytokine production by immune lymphocytes in herpes simplex virus-infected mice.lld:pubmed
pubmed-article:7865481pubmed:affiliationDepartment of Dermatology, Faculty of Medicine, Kyushu University, Fukuoka, Japan.lld:pubmed
pubmed-article:7865481pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:7865481pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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