| pubmed-article:7783619 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7783619 | lifeskim:mentions | umls-concept:C0085455 | lld:lifeskim |
| pubmed-article:7783619 | lifeskim:mentions | umls-concept:C1457869 | lld:lifeskim |
| pubmed-article:7783619 | lifeskim:mentions | umls-concept:C0038855 | lld:lifeskim |
| pubmed-article:7783619 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:7783619 | pubmed:dateCreated | 1995-7-19 | lld:pubmed |
| pubmed-article:7783619 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:abstractText | An insertion of transposon Tn5-lac, omega 4519, generates a lacZ fusion with a Myxococcus xanthus promoter expressed during both vegetative growth and development. Sequence analysis of the junction of omega 4519 with M. xanthus DNA shows that the insertion is in frzF, a homologue of cheR from Salmonella typhimurium. When frzF- (or frzCD-) cells are starved for nutrients at modest densities, they aggregate to form a radial pattern and produce fewer than 1% of the wild-type complement of spores. At higher densities, frzF::omega 4519 cells form 'frizzy' aggregates and produce 80-90% of the wild-type complement of spores. In contrast, when cells with both a frzF- (or frzCD-) and an sglA1 mutation are allowed to develop at either low or high cell densities, they produce frizzy aggregates containing a near wild-type complement of heat-resistant spores. In addition to suppressing the density dependence of fruiting-body morphogenesis, the sglA1 mutation also suppresses the sporulation defect caused by two different frzF- mutations and a frzCD- mutation. In contrast, a mutation in a different S motility gene, sglG1, does not suppress the frz- mutations. Thus, the suppression of frz- mutations by sgl- mutations is allele-specific, and depends on the sgl allele, but not the frz allele. Because the phenotypes of frz- mutations have been determined in a (suppressing) sglA1 genetic background, the frz genes may play more central roles in development than initially recognized. | lld:pubmed |
| pubmed-article:7783619 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7783619 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7783619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783619 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7783619 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:7783619 | pubmed:issn | 0950-382X | lld:pubmed |
| pubmed-article:7783619 | pubmed:author | pubmed-author:HartzellP LPL | lld:pubmed |
| pubmed-article:7783619 | pubmed:author | pubmed-author:KashefiKK | lld:pubmed |
| pubmed-article:7783619 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7783619 | pubmed:volume | 15 | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | cheR | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | sgl | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | agl | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | cgl | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | mglA | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | frzF | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | bsgA | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | frz | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | frzC | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | frzD | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | aglB1 | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | sglA1 | lld:pubmed |
| pubmed-article:7783619 | pubmed:geneSymbol | sglG1 | lld:pubmed |
| pubmed-article:7783619 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7783619 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7783619 | pubmed:pagination | 483-94 | lld:pubmed |
| pubmed-article:7783619 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:7783619 | pubmed:meshHeading | pubmed-meshheading:7783619-... | lld:pubmed |
| pubmed-article:7783619 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7783619 | pubmed:articleTitle | Genetic suppression and phenotypic masking of a Myxococcus xanthus frzF- defect. | lld:pubmed |
| pubmed-article:7783619 | pubmed:affiliation | Department of Microbiology and Molecular Genetics, University of California, Los Angeles 90024-1489, USA. | lld:pubmed |
| pubmed-article:7783619 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7783619 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:7783619 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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