pubmed-article:7660337 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:7660337 | lifeskim:mentions | umls-concept:C0004611 | lld:lifeskim |
pubmed-article:7660337 | lifeskim:mentions | umls-concept:C0024117 | lld:lifeskim |
pubmed-article:7660337 | lifeskim:mentions | umls-concept:C0080103 | lld:lifeskim |
pubmed-article:7660337 | lifeskim:mentions | umls-concept:C0597979 | lld:lifeskim |
pubmed-article:7660337 | lifeskim:mentions | umls-concept:C0815172 | lld:lifeskim |
pubmed-article:7660337 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:7660337 | pubmed:dateCreated | 1995-9-29 | lld:pubmed |
pubmed-article:7660337 | pubmed:abstractText | In addition to bronchodilator and anti-inflammatory therapy, exacerbations in patients with chronic obstructive pulmonary disease (COPD) are often treated with antibiotics. Haemophilus influenzae and Moraxella (Branhamella) catarrhalis, two important respiratory pathogens, may produce beta-lactamase which makes them resistant to ampicillin. Surveillance studies conducted in various countries have shown an increasing incidence of these beta-lactamase producing bacteria. Although this may simply be a consequence of the increasing use of antibiotics, it is possible that other factors are important. A study was undertaken to investigate whether clinical factors are related to the presence of beta-lactamase forming bacteria in the sputum of patients with COPD. | lld:pubmed |
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pubmed-article:7660337 | pubmed:language | eng | lld:pubmed |
pubmed-article:7660337 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7660337 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:7660337 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7660337 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:7660337 | pubmed:month | Mar | lld:pubmed |
pubmed-article:7660337 | pubmed:issn | 0040-6376 | lld:pubmed |
pubmed-article:7660337 | pubmed:author | pubmed-author:LöwenbergAA | lld:pubmed |
pubmed-article:7660337 | pubmed:author | pubmed-author:KoëterG HGH | lld:pubmed |
pubmed-article:7660337 | pubmed:author | pubmed-author:van AltenaRR | lld:pubmed |
pubmed-article:7660337 | pubmed:author | pubmed-author:BoersmaW GWG | lld:pubmed |
pubmed-article:7660337 | pubmed:author | pubmed-author:SportelJ HJH | lld:pubmed |
pubmed-article:7660337 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:7660337 | pubmed:volume | 50 | lld:pubmed |
pubmed-article:7660337 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:7660337 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:7660337 | pubmed:pagination | 249-53 | lld:pubmed |
pubmed-article:7660337 | pubmed:dateRevised | 2010-3-24 | lld:pubmed |
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pubmed-article:7660337 | pubmed:year | 1995 | lld:pubmed |
pubmed-article:7660337 | pubmed:articleTitle | Relation between beta-lactamase producing bacteria and patient characteristics in chronic obstructive pulmonary disease (COPD). | lld:pubmed |
pubmed-article:7660337 | pubmed:affiliation | Department of Pulmonary Diseases, University Hospital, Groningen, The Netherlands. | lld:pubmed |
pubmed-article:7660337 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:7660337 | pubmed:publicationType | Comparative Study | lld:pubmed |
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