pubmed-article:7043093 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:7043093 | lifeskim:mentions | umls-concept:C0450442 | lld:lifeskim |
pubmed-article:7043093 | lifeskim:mentions | umls-concept:C0045052 | lld:lifeskim |
pubmed-article:7043093 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:7043093 | pubmed:dateCreated | 1982-7-19 | lld:pubmed |
pubmed-article:7043093 | pubmed:abstractText | The hypertensiogenic effects of 19-hydroxyandrostenedione (19-OH-A-dione) reported previously to be an amplifier of the action of aldosterone were evaluated using the same experimental procedures as for DOCA-salt hypertension in rats. The 19-OH-A-dione treated rats developed high blood pressure, hypokalemia, suppressed plasma renin activity and low plasma aldosterone and corticosterone concentrations as the DOCA treated rats did. The results demonstrate that 19-OH-A-dione amplifies the action of endogenous aldosterone and causes a hypertensive state simulating mineralocorticoid excess. Amplifiers of the action of aldosterone are considered to work as hypertensinogenic agents in the presence of the adrenal cortex. | lld:pubmed |
pubmed-article:7043093 | pubmed:language | eng | lld:pubmed |
pubmed-article:7043093 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7043093 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:7043093 | pubmed:month | Feb | lld:pubmed |
pubmed-article:7043093 | pubmed:issn | 0022-4731 | lld:pubmed |
pubmed-article:7043093 | pubmed:author | pubmed-author:SekiharaHH | lld:pubmed |
pubmed-article:7043093 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:7043093 | pubmed:volume | 16 | lld:pubmed |
pubmed-article:7043093 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:7043093 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:7043093 | pubmed:pagination | 329-31 | lld:pubmed |
pubmed-article:7043093 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:7043093 | pubmed:meshHeading | pubmed-meshheading:7043093-... | lld:pubmed |
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pubmed-article:7043093 | pubmed:meshHeading | pubmed-meshheading:7043093-... | lld:pubmed |
pubmed-article:7043093 | pubmed:year | 1982 | lld:pubmed |
pubmed-article:7043093 | pubmed:articleTitle | 19-hydroxyandrostenedione as a new hypertensinogenic agent. | lld:pubmed |
pubmed-article:7043093 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:7043093 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:7043093 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:7043093 | lld:pubmed |