| pubmed-article:6308123 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0020663 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0001473 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0175630 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0449416 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C1999216 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
| pubmed-article:6308123 | lifeskim:mentions | umls-concept:C0332120 | lld:lifeskim |
| pubmed-article:6308123 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:6308123 | pubmed:dateCreated | 1983-9-20 | lld:pubmed |
| pubmed-article:6308123 | pubmed:abstractText | Acetone extracts from a variety of rat tissues were tested for their ability to stimulate renal glucose-6-phosphate dehydrogenase (G6PD) activity at 2 min in an in-vitro cytochemical assay which is a marker of the sodium potassium-dependent adenosine triphosphatase (Na+-K+-ATPase) inhibiting activity. Extracts of the hypothalamus were the only ones found to be active in this system. Acetone extract of hypothalamus also inhibited renal Na+-K+-ATPase activity in vitro. The G6PD-stimulating activity from one hypothalamus was about 10000 to 100000 times greater than that of 1 ml plasma. The G6PD-stimulating activity of hypothalamic extracts from rats which had been on a high sodium intake for 4 weeks were approximately 150 times more active than those obtained from rats which had been on a low sodium diet. The G6PD-stimulating activity of the corresponding plasma was sixfold more active. These findings suggest that a circulating sodium transport inhibitor(s) may be secreted from the hypothalamus. | lld:pubmed |
| pubmed-article:6308123 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6308123 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6308123 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:6308123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6308123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6308123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6308123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6308123 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6308123 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:6308123 | pubmed:issn | 0022-0795 | lld:pubmed |
| pubmed-article:6308123 | pubmed:author | pubmed-author:de... | lld:pubmed |
| pubmed-article:6308123 | pubmed:author | pubmed-author:Alaghband-Zad... | lld:pubmed |
| pubmed-article:6308123 | pubmed:author | pubmed-author:HancockKK | lld:pubmed |
| pubmed-article:6308123 | pubmed:author | pubmed-author:FentonSS | lld:pubmed |
| pubmed-article:6308123 | pubmed:author | pubmed-author:MillettJJ | lld:pubmed |
| pubmed-article:6308123 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6308123 | pubmed:volume | 98 | lld:pubmed |
| pubmed-article:6308123 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6308123 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6308123 | pubmed:pagination | 221-6 | lld:pubmed |
| pubmed-article:6308123 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:meshHeading | pubmed-meshheading:6308123-... | lld:pubmed |
| pubmed-article:6308123 | pubmed:year | 1983 | lld:pubmed |
| pubmed-article:6308123 | pubmed:articleTitle | Evidence that the hypothalamus may be a source of a circulating Na+-K+-ATPase inhibitor. | lld:pubmed |
| pubmed-article:6308123 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6308123 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
