pubmed-article:6281743 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6281743 | lifeskim:mentions | umls-concept:C0341628 | lld:lifeskim |
pubmed-article:6281743 | lifeskim:mentions | umls-concept:C0229671 | lld:lifeskim |
pubmed-article:6281743 | lifeskim:mentions | umls-concept:C0040135 | lld:lifeskim |
pubmed-article:6281743 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
pubmed-article:6281743 | lifeskim:mentions | umls-concept:C1261322 | lld:lifeskim |
pubmed-article:6281743 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:6281743 | pubmed:dateCreated | 1982-7-8 | lld:pubmed |
pubmed-article:6281743 | pubmed:abstractText | Intravascular application of heparin causes a significant rise of free and total thyroid hormone concentrations in serum together with a concomitant drop of the TSH-levels. A causal relationship between these two findings has been firmly established by a number of investigators. The decline of TSH concentrations in serum after heparin injection might just be a physiological reaction of the pituitary gland to the heparin-triggered rise of serum thyroid hormone levels. The present investigations demonstrate a diminution of affinity of thyroid hormone receptors of liver cells. This result confirms the hypothesis that the rise of total thyroid hormone in blood is mainly due to a depletion of liver stores of thyroid hormones. In concordance with clinical findings the binding affinity of cellular pituitary thyroid hormone receptors was found to increase. | lld:pubmed |
pubmed-article:6281743 | pubmed:language | ger | lld:pubmed |
pubmed-article:6281743 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6281743 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:6281743 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6281743 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6281743 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6281743 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6281743 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6281743 | pubmed:month | Feb | lld:pubmed |
pubmed-article:6281743 | pubmed:issn | 0029-5566 | lld:pubmed |
pubmed-article:6281743 | pubmed:author | pubmed-author:BeyerH KHK | lld:pubmed |
pubmed-article:6281743 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6281743 | pubmed:volume | 21 | lld:pubmed |
pubmed-article:6281743 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6281743 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6281743 | pubmed:pagination | 36-41 | lld:pubmed |
pubmed-article:6281743 | pubmed:dateRevised | 2007-1-12 | lld:pubmed |
pubmed-article:6281743 | pubmed:meshHeading | pubmed-meshheading:6281743-... | lld:pubmed |
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pubmed-article:6281743 | pubmed:meshHeading | pubmed-meshheading:6281743-... | lld:pubmed |
pubmed-article:6281743 | pubmed:year | 1982 | lld:pubmed |
pubmed-article:6281743 | pubmed:articleTitle | [Causative investigations of heparin-induced rise on thyroid hormone serum levels (author's transl)]. | lld:pubmed |
pubmed-article:6281743 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:6281743 | pubmed:publicationType | In Vitro | lld:pubmed |
pubmed-article:6281743 | pubmed:publicationType | English Abstract | lld:pubmed |