| pubmed-article:6086315 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:6086315 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:6086315 | lifeskim:mentions | umls-concept:C0027260 | lld:lifeskim |
| pubmed-article:6086315 | lifeskim:mentions | umls-concept:C0016057 | lld:lifeskim |
| pubmed-article:6086315 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:6086315 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:6086315 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:6086315 | pubmed:dateCreated | 1984-8-24 | lld:pubmed |
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| pubmed-article:6086315 | pubmed:abstractText | A full length N-ras gene has been cloned from both the human fibrosarcoma cell line HT1080 and from normal human DNA. N-ras isolated from HT1080 will efficiently induce morphological transformation of NIH/3T3 cells in a transfection assay, whereas N-ras isolated from normal human DNA has no effect on NIH/3T3 cells. The coding regions of the normal N-ras gene have been sequenced and the predicted amino acid sequence of the N-ras product is very similar to that of the c-Ha-ras1 and c-Ki-ras2 products. By making chimeric molecules between the two cloned genes the activating alteration in the HT1080 N-ras gene has been localised to a single base change that results in an amino acid alteration at position 61 of the p21 N-ras product. | lld:pubmed |
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| pubmed-article:6086315 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:6086315 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6086315 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6086315 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:6086315 | pubmed:issn | 0261-4189 | lld:pubmed |
| pubmed-article:6086315 | pubmed:author | pubmed-author:MarshallC JCJ | lld:pubmed |
| pubmed-article:6086315 | pubmed:author | pubmed-author:BrownRR | lld:pubmed |
| pubmed-article:6086315 | pubmed:author | pubmed-author:HallAA | lld:pubmed |
| pubmed-article:6086315 | pubmed:author | pubmed-author:PennieS GSG | lld:pubmed |
| pubmed-article:6086315 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6086315 | pubmed:volume | 3 | lld:pubmed |
| pubmed-article:6086315 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6086315 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6086315 | pubmed:pagination | 1321-6 | lld:pubmed |
| pubmed-article:6086315 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:6086315 | pubmed:year | 1984 | lld:pubmed |
| pubmed-article:6086315 | pubmed:articleTitle | Mechanism of activation of an N-ras gene in the human fibrosarcoma cell line HT1080. | lld:pubmed |
| pubmed-article:6086315 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6086315 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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