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pubmed-article:3689372pubmed:abstractTextPhenothiazines at high concentrations inhibit platelet aggregation and the secretion of granule contents. In this study we have evaluated the influence of stelazine on platelet function at low concentrations. Stelazine alone had no influence on resting calcium levels in platelets but facilitated agonist-induced elevation of cytosolic calcium. Platelets combined with low concentrations of stelazine (10 microM) and stimulated with subthreshold concentrations of thrombin (0.05 mu/ml) aggregated irreversibly and released significant quantities of ATP. Results of these studies suggest a new role for the calmodulin antagonist stelazine in platelet activation.lld:pubmed
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pubmed-article:3689372pubmed:articleTitleInfluence of calmodulin antagonist (stelazine) on agonist-induced calcium mobilization and platelet activation.lld:pubmed
pubmed-article:3689372pubmed:affiliationDepartment of Laboratory Medicine and Pathology, University of Minnesota Health Sciences Center, Minneapolis 55455.lld:pubmed
pubmed-article:3689372pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:3689372pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
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