| pubmed-article:3484651 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0007776 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0022655 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0001613 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0740279 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C2348519 | lld:lifeskim |
| pubmed-article:3484651 | lifeskim:mentions | umls-concept:C0205087 | lld:lifeskim |
| pubmed-article:3484651 | pubmed:dateCreated | 1986-3-13 | lld:pubmed |
| pubmed-article:3484651 | pubmed:abstractText | Ten patients with late cortical cerebellar atrophy documented with computerized tomography (CT) and magnetic resonance (MR) imaging presented a uniform pattern of oculomotor abnormalities. All had deficits in smooth pursuit and fixation suppression of the vestibulo-ocular reflex (VOR). The gain of the VOR was often increased but the dynamics of the VOR were not different from normals. Full-field optokinetic responses and visual-vestibular interaction mediated via the VOR velocity storage element were preserved. Just as in lower primates, the cerebellum in humans is a critical relay station for foveal pursuit and visual-vestibular interaction requiring foveal pursuit. Vestibular responses and visual-vestibular interaction mediated through the VOR velocity storage element are relatively independent of the cerebellum. | lld:pubmed |
| pubmed-article:3484651 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3484651 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3484651 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3484651 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3484651 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3484651 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:3484651 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3484651 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:3484651 | pubmed:issn | 0006-8950 | lld:pubmed |
| pubmed-article:3484651 | pubmed:author | pubmed-author:BalohR WRW | lld:pubmed |
| pubmed-article:3484651 | pubmed:author | pubmed-author:HonrubiaVV | lld:pubmed |
| pubmed-article:3484651 | pubmed:author | pubmed-author:YeeR DRD | lld:pubmed |
| pubmed-article:3484651 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3484651 | pubmed:volume | 109 ( Pt 1) | lld:pubmed |
| pubmed-article:3484651 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3484651 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3484651 | pubmed:pagination | 159-80 | lld:pubmed |
| pubmed-article:3484651 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:3484651 | pubmed:meshHeading | pubmed-meshheading:3484651-... | lld:pubmed |
| pubmed-article:3484651 | pubmed:meshHeading | pubmed-meshheading:3484651-... | lld:pubmed |
| pubmed-article:3484651 | pubmed:meshHeading | pubmed-meshheading:3484651-... | lld:pubmed |
| pubmed-article:3484651 | pubmed:meshHeading | pubmed-meshheading:3484651-... | lld:pubmed |
| pubmed-article:3484651 | pubmed:meshHeading | pubmed-meshheading:3484651-... | lld:pubmed |
| pubmed-article:3484651 | pubmed:year | 1986 | lld:pubmed |
| pubmed-article:3484651 | pubmed:articleTitle | Late cortical cerebellar atrophy. Clinical and oculographic features. | lld:pubmed |
| pubmed-article:3484651 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3484651 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3484651 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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