pubmed-article:3043188 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3043188 | lifeskim:mentions | umls-concept:C0015576 | lld:lifeskim |
pubmed-article:3043188 | lifeskim:mentions | umls-concept:C0680022 | lld:lifeskim |
pubmed-article:3043188 | lifeskim:mentions | umls-concept:C0017287 | lld:lifeskim |
pubmed-article:3043188 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
pubmed-article:3043188 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:3043188 | pubmed:dateCreated | 1988-9-16 | lld:pubmed |
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pubmed-article:3043188 | pubmed:abstractText | Complementary DNA clones of a putative transforming gene were isolated from NIH 3T3 cells transformed with human Ewing sarcoma DNA. The gene was termed B-raf because it is related to but distinct from c-raf and A-raf. It appears that substitution in the amino-terminal portion of the normal B-raf protein confers transforming activity to the gene. | lld:pubmed |
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pubmed-article:3043188 | pubmed:language | eng | lld:pubmed |
pubmed-article:3043188 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3043188 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:3043188 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3043188 | pubmed:month | Jun | lld:pubmed |
pubmed-article:3043188 | pubmed:issn | 0270-7306 | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:YamamotoTT | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:IkawaSS | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:ToyoshimaKK | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:FukuiMM | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:TamaokiNN | lld:pubmed |
pubmed-article:3043188 | pubmed:author | pubmed-author:UeyamaYY | lld:pubmed |
pubmed-article:3043188 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3043188 | pubmed:volume | 8 | lld:pubmed |
pubmed-article:3043188 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3043188 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3043188 | pubmed:pagination | 2651-4 | lld:pubmed |
pubmed-article:3043188 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:3043188 | pubmed:year | 1988 | lld:pubmed |
pubmed-article:3043188 | pubmed:articleTitle | B-raf, a new member of the raf family, is activated by DNA rearrangement. | lld:pubmed |
pubmed-article:3043188 | pubmed:affiliation | Department of Oncology, University of Tokyo, Japan. | lld:pubmed |
pubmed-article:3043188 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:3043188 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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