| pubmed-article:2960665 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C0926407 | lld:lifeskim |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C0162326 | lld:lifeskim |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:2960665 | lifeskim:mentions | umls-concept:C1527180 | lld:lifeskim |
| pubmed-article:2960665 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:2960665 | pubmed:dateCreated | 1988-1-14 | lld:pubmed |
| pubmed-article:2960665 | pubmed:abstractText | We have combined techniques of genetic and physical mapping with rapid DNA sequence analysis to identify the nucleotide change in lambda int mutations. These mutations define two dominant phenotypic classes: (i) recombination that is partially independent of accessory factors, and (ii) inhibition of wild-type Int by missense or nonsense proteins, i.e., negative complementation. | lld:pubmed |
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| pubmed-article:2960665 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2960665 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2960665 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2960665 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2960665 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2960665 | pubmed:issn | 0021-9193 | lld:pubmed |
| pubmed-article:2960665 | pubmed:author | pubmed-author:EnquistL WLW | lld:pubmed |
| pubmed-article:2960665 | pubmed:author | pubmed-author:BearS ESE | lld:pubmed |
| pubmed-article:2960665 | pubmed:author | pubmed-author:ClemensJ BJB | lld:pubmed |
| pubmed-article:2960665 | pubmed:author | pubmed-author:ZagurskyR JRJ | lld:pubmed |
| pubmed-article:2960665 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2960665 | pubmed:volume | 169 | lld:pubmed |
| pubmed-article:2960665 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2960665 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2960665 | pubmed:pagination | 5880-3 | lld:pubmed |
| pubmed-article:2960665 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:2960665 | pubmed:meshHeading | pubmed-meshheading:2960665-... | lld:pubmed |
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| pubmed-article:2960665 | pubmed:meshHeading | pubmed-meshheading:2960665-... | lld:pubmed |
| pubmed-article:2960665 | pubmed:meshHeading | pubmed-meshheading:2960665-... | lld:pubmed |
| pubmed-article:2960665 | pubmed:meshHeading | pubmed-meshheading:2960665-... | lld:pubmed |
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| pubmed-article:2960665 | pubmed:meshHeading | pubmed-meshheading:2960665-... | lld:pubmed |
| pubmed-article:2960665 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:2960665 | pubmed:articleTitle | Mutational analysis of the lambda int gene: DNA sequence of dominant mutations. | lld:pubmed |
| pubmed-article:2960665 | pubmed:affiliation | Department of Molecular Biology, E. I. du Pont de Nemours & Co., Inc., Wilmington, Delaware 19898. | lld:pubmed |
| pubmed-article:2960665 | pubmed:publicationType | Journal Article | lld:pubmed |
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