| pubmed-article:2853731 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2853731 | lifeskim:mentions | umls-concept:C0020538 | lld:lifeskim |
| pubmed-article:2853731 | lifeskim:mentions | umls-concept:C0006726 | lld:lifeskim |
| pubmed-article:2853731 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:2853731 | lifeskim:mentions | umls-concept:C1159651 | lld:lifeskim |
| pubmed-article:2853731 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
| pubmed-article:2853731 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:2853731 | pubmed:dateCreated | 1989-5-18 | lld:pubmed |
| pubmed-article:2853731 | pubmed:abstractText | Erythrocyte cation transport and intracellular calcium in 15 black type II diabetic hypertensives were compared with 11 otherwise similar non-diabetic hypertensives and 16 normal black adults. The diabetic hypertensives were then randomized into either a placebo group or a calcium-supplemented (600 mg/day) group and studied again after 4 weeks. Na+,K+-ATPase activity was significantly lower in both groups of hypertensives than in the normotensives. In contrast, Ca-ATPase activity was similar among the non-diabetic hypertensive and normotensives but was markedly (approximately 60%) suppressed in the diabetics, while intracellular calcium was proportionally elevated. Calcium supplements significantly increased Ca-ATPase activity and reduced intracellular calcium and blood pressure compared with the placebo group. We conclude that type II diabetes is characterized by a defect in Ca-ATPase which may be responsible for increases in intracellular calcium and vascular resistance and which is partially corrected by dietary calcium supplementation. | lld:pubmed |
| pubmed-article:2853731 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2853731 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2853731 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2853731 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2853731 | pubmed:issn | 0952-1178 | lld:pubmed |
| pubmed-article:2853731 | pubmed:author | pubmed-author:SowersJ RJR | lld:pubmed |
| pubmed-article:2853731 | pubmed:author | pubmed-author:ZemelP CPC | lld:pubmed |
| pubmed-article:2853731 | pubmed:author | pubmed-author:ZemelM BMB | lld:pubmed |
| pubmed-article:2853731 | pubmed:author | pubmed-author:BedfordB ABA | lld:pubmed |
| pubmed-article:2853731 | pubmed:author | pubmed-author:MarwahOO | lld:pubmed |
| pubmed-article:2853731 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2853731 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:2853731 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2853731 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2853731 | pubmed:pagination | S228-30 | lld:pubmed |
| pubmed-article:2853731 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:meshHeading | pubmed-meshheading:2853731-... | lld:pubmed |
| pubmed-article:2853731 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:2853731 | pubmed:articleTitle | Altered cation transport in non-insulin-dependent diabetic hypertension: effects of dietary calcium. | lld:pubmed |
| pubmed-article:2853731 | pubmed:affiliation | Division of Endocrinology, Metabolism and Hypertension, Wayne State University, Detroit, Michigan 48202. | lld:pubmed |
| pubmed-article:2853731 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2853731 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:2853731 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:2853731 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:2853731 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2853731 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2853731 | lld:pubmed |
