pubmed-article:2787191 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0003826 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0003250 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0034819 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0027814 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C1412286 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0599894 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
pubmed-article:2787191 | lifeskim:mentions | umls-concept:C0301625 | lld:lifeskim |
pubmed-article:2787191 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:2787191 | pubmed:dateCreated | 1989-8-21 | lld:pubmed |
pubmed-article:2787191 | pubmed:abstractText | The monoclonal antibody ART 18 directed to the rat interleukin-2 receptor (IL-2 R) was administered to Lewis rats immediately prior to and/or on consecutive days after adoptive transfer of autoreactive P2-T line lymphocytes. The effects of ART 18 and sham treatment on the development of adoptive transfer--experimental autoimmune neuritis (AT-EAN) were assessed by clinical inspection, serial electrophysiological monitoring, and semiquantitative histomorphological analysis. Early injection of ART 18 suppressed AT-EAN while treatment after appearance of clinical signs did not. Since the IL-2 R is expressed exclusively on proliferating T cells activated by antigen, the in vivo application of an IL-2 R-targeted monoclonal antibody allows for more selective immunosuppression of experimental autoimmune disease of the peripheral nervous system than has previously been achieved. | lld:pubmed |
pubmed-article:2787191 | pubmed:language | eng | lld:pubmed |
pubmed-article:2787191 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2787191 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:2787191 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2787191 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2787191 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2787191 | pubmed:month | Jun | lld:pubmed |
pubmed-article:2787191 | pubmed:issn | 0006-8993 | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:HeiningerKK | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:SchäferBB | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:ToykaK VKV | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:FierzWW | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:DiamantsteinT... | lld:pubmed |
pubmed-article:2787191 | pubmed:author | pubmed-author:HartungH PHP | lld:pubmed |
pubmed-article:2787191 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2787191 | pubmed:day | 5 | lld:pubmed |
pubmed-article:2787191 | pubmed:volume | 489 | lld:pubmed |
pubmed-article:2787191 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2787191 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2787191 | pubmed:pagination | 120-8 | lld:pubmed |
pubmed-article:2787191 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:meshHeading | pubmed-meshheading:2787191-... | lld:pubmed |
pubmed-article:2787191 | pubmed:year | 1989 | lld:pubmed |
pubmed-article:2787191 | pubmed:articleTitle | Suppression of P2-T cell line-mediated experimental autoimmune neuritis by interleukin-2 receptor targeted monoclonal antibody ART 18. | lld:pubmed |
pubmed-article:2787191 | pubmed:affiliation | Department of Neurology, University of Düsseldorf, F.R.G. | lld:pubmed |
pubmed-article:2787191 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2787191 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2787191 | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2787191 | lld:pubmed |