| pubmed-article:2595271 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0013303 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0022378 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0017128 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0268009 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C1522821 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C1160577 | lld:lifeskim |
| pubmed-article:2595271 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
| pubmed-article:2595271 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:2595271 | pubmed:dateCreated | 1990-1-17 | lld:pubmed |
| pubmed-article:2595271 | pubmed:abstractText | In chronic gastric fistula (GF) rats, HCl and a hyperosmolal solution of polyethylene glycol (PEG) in the upper intestine inhibit pentagastrin-stimulated gastric acid secretion by different mechanisms, but their anatomic sites have not yet been established. In the present study GF rats were provided with Thiry-Vella loops of the duodenum and the bile and pancreatic ducts transplanted to the proximal jejunum, or with Thiry-Vella loops of the proximal jejunum. In the latter rats the duodenum was anastomosed as a blind loop to the jejunum to prevent any gastric juice from entering the duodenum. Duodenal loop perfusion with 0.20 M HCl inhibited the acid response to pentagastrin by 62%, but perfusion with 1200 mOsmol x kg-1 of PEG solution did not alter the response. In contrast, acidification of the proximal jejunal loop did not alter but hyperosmolality inhibited the response by 41%. The study shows that the mechanism for inhibition by intestinal acidification is confined to the duodenum and that for inhibition by hyperosmolality is located in the proximal jejunum--but whether only to the proximal part is unknown. | lld:pubmed |
| pubmed-article:2595271 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2595271 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2595271 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2595271 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2595271 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2595271 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2595271 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2595271 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:2595271 | pubmed:issn | 0036-5521 | lld:pubmed |
| pubmed-article:2595271 | pubmed:author | pubmed-author:EmåsSS | lld:pubmed |
| pubmed-article:2595271 | pubmed:author | pubmed-author:WallinCC | lld:pubmed |
| pubmed-article:2595271 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2595271 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:2595271 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2595271 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2595271 | pubmed:pagination | 1095-101 | lld:pubmed |
| pubmed-article:2595271 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:meshHeading | pubmed-meshheading:2595271-... | lld:pubmed |
| pubmed-article:2595271 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2595271 | pubmed:articleTitle | Duodenal acidification and jejunal hyperosmolality inhibit pentagastrin-stimulated acid secretion in chronic gastric fistula rats. | lld:pubmed |
| pubmed-article:2595271 | pubmed:affiliation | Dept. of Surgery, Karolinska Hospital, Stockholm, Sweden. | lld:pubmed |
| pubmed-article:2595271 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2595271 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
