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| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C0032136 | lld:lifeskim |
| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C1510411 | lld:lifeskim |
| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C1257741 | lld:lifeskim |
| pubmed-article:2398894 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:2398894 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:2398894 | pubmed:dateCreated | 1990-10-18 | lld:pubmed |
| pubmed-article:2398894 | pubmed:abstractText | The enterobacterial plasmid misrepair gene mucAB, ligated to the metal-inducible mammalian MT-1 promoter, was introduced into the genome of mouse BALB 3T3 cells. In the presence of zinc ions, MucA but not MucB protein was produced, and the whole-cell population of each mucAB+ clone started to show the transformation phenotype in a few days. Foci appeared in the transformed cell population after 4 weeks, and cells from the foci produced tumors in nude mice, indicating malignant transformation by the mucA product. Growth of mucAB+ cells was stimulated by zinc-induced expression of mucA. The transformation phenotype was reversed by removing zinc ions from the culture, indicating that the transformation was due not to MucA-mediated mutation in the mouse genome but to the direct transforming activity of MucA protein. | lld:pubmed |
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| pubmed-article:2398894 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2398894 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2398894 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2398894 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2398894 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:2398894 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:2398894 | pubmed:author | pubmed-author:TanookaHH | lld:pubmed |
| pubmed-article:2398894 | pubmed:author | pubmed-author:TosoGG | lld:pubmed |
| pubmed-article:2398894 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2398894 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:2398894 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2398894 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2398894 | pubmed:pagination | 5359-64 | lld:pubmed |
| pubmed-article:2398894 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2398894 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2398894 | pubmed:articleTitle | Transformation of mouse BALB 3T3 cells by enterobacterial plasmid misrepair gene mucAB. | lld:pubmed |
| pubmed-article:2398894 | pubmed:affiliation | Radiobiology Division, National Cancer Center Research Institute, Tokyo, Japan. | lld:pubmed |
| pubmed-article:2398894 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2398894 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:2398894 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2398894 | lld:pubmed |
