pubmed-article:21698265 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C0032043 | lld:lifeskim |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C0743925 | lld:lifeskim |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
pubmed-article:21698265 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
pubmed-article:21698265 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:21698265 | pubmed:dateCreated | 2011-6-23 | lld:pubmed |
pubmed-article:21698265 | pubmed:abstractText | Novel research has suggested that altered miRNA expression in the placenta is associated with adverse pregnancy outcomes and with potentially harmful xenobiotic exposures. We hypothesized that aberrant expression of miRNA in the placenta is associated with fetal growth, a measurable phenotype resulting from a number of intrauterine factors, and one which is significantly predictive of later life outcomes. | lld:pubmed |
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pubmed-article:21698265 | pubmed:language | eng | lld:pubmed |
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pubmed-article:21698265 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:21698265 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21698265 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:21698265 | pubmed:author | pubmed-author:PadburyJames... | lld:pubmed |
pubmed-article:21698265 | pubmed:author | pubmed-author:MarsitCarmen... | lld:pubmed |
pubmed-article:21698265 | pubmed:author | pubmed-author:MaccaniMatthe... | lld:pubmed |
pubmed-article:21698265 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21698265 | pubmed:volume | 6 | lld:pubmed |
pubmed-article:21698265 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21698265 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21698265 | pubmed:pagination | e21210 | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:meshHeading | pubmed-meshheading:21698265... | lld:pubmed |
pubmed-article:21698265 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21698265 | pubmed:articleTitle | miR-16 and miR-21 expression in the placenta is associated with fetal growth. | lld:pubmed |
pubmed-article:21698265 | pubmed:affiliation | Department of Pathology and Laboratory Medicine, Brown University, Providence, Rhode Island, United States of America. | lld:pubmed |
pubmed-article:21698265 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21698265 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
entrez-gene:406991 | entrezgene:pubmed | pubmed-article:21698265 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:21698265 | lld:entrezgene |