| pubmed-article:21697995 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C1148798 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C0239059 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C1155342 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C1522240 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C1512488 | lld:lifeskim |
| pubmed-article:21697995 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:21697995 | pubmed:dateCreated | 2011-6-23 | lld:pubmed |
| pubmed-article:21697995 | pubmed:abstractText | Cigarette smoke is the major risk factor associated with the development of chronic obstructive pulmonary disease (COPD). Recent studies propose a link between endoplasmic reticulum (ER) stress and emphysema, demonstrated by increased ER stress markers under smoking conditions. Here, we investigate whether cigarette smoke-induced ER stress is cell specific and correlates with acute and chronic cigarette smoke exposure. | lld:pubmed |
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| pubmed-article:21697995 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21697995 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21697995 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21697995 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21697995 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:21697995 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21697995 | pubmed:issn | 1178-2005 | lld:pubmed |
| pubmed-article:21697995 | pubmed:author | pubmed-author:GeraghtyPatri... | lld:pubmed |
| pubmed-article:21697995 | pubmed:author | pubmed-author:WallaceAlison... | lld:pubmed |
| pubmed-article:21697995 | pubmed:author | pubmed-author:D'ArmientoJea... | lld:pubmed |
| pubmed-article:21697995 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21697995 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:21697995 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21697995 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21697995 | pubmed:pagination | 309-19 | lld:pubmed |
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| pubmed-article:21697995 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21697995 | pubmed:articleTitle | Induction of the unfolded protein response by cigarette smoke is primarily an activating transcription factor 4-C/EBP homologous protein mediated process. | lld:pubmed |
| pubmed-article:21697995 | pubmed:affiliation | Department of Medicine, Divisions of Molecular and Pulmonary Medicine, Columbia University College of Physicians and Surgeons, New York, NY 10032, USA. | lld:pubmed |
| pubmed-article:21697995 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21697995 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| entrez-gene:1649 | entrezgene:pubmed | pubmed-article:21697995 | lld:entrezgene |
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