| pubmed-article:21642892 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C1956346 | lld:lifeskim |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C1414433 | lld:lifeskim |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C1413880 | lld:lifeskim |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:21642892 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:21642892 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:21642892 | pubmed:dateCreated | 2011-7-12 | lld:pubmed |
| pubmed-article:21642892 | pubmed:abstractText | Common polymorphisms within cytochrome P450 2J2 (CYP2J2) and epoxide hydrolase 2 (EPHX2), which are involved in the generation or hydrolysis of epoxyeicosatrienoic acids, may determine susceptibility to the development of cardiovascular disease. To derive a more precise estimation of their relationship, we undertook a case-control study as well as a meta-analysis to assess possible associations of coronary artery disease (CAD) risk with CYP2J2 and EPHX2 genetic variations. | lld:pubmed |
| pubmed-article:21642892 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21642892 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21642892 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21642892 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:21642892 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21642892 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:21642892 | pubmed:issn | 1744-6880 | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:RAOT TTT | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:WangDao WenDW | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:CianfloneKath... | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:DingHuH | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:CuiGuanglinG | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:PengJiaJ | lld:pubmed |
| pubmed-article:21642892 | pubmed:author | pubmed-author:XuYujunY | lld:pubmed |
| pubmed-article:21642892 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21642892 | pubmed:volume | 21 | lld:pubmed |
| pubmed-article:21642892 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21642892 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21642892 | pubmed:pagination | 489-94 | lld:pubmed |
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| pubmed-article:21642892 | pubmed:meshHeading | pubmed-meshheading:21642892... | lld:pubmed |
| pubmed-article:21642892 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21642892 | pubmed:articleTitle | Association between polymorphisms of CYP2J2 and EPHX2 genes and risk of coronary artery disease. | lld:pubmed |
| pubmed-article:21642892 | pubmed:affiliation | The Institute of Hypertension and Department of Internal Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China. | lld:pubmed |
| pubmed-article:21642892 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21642892 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:21642892 | pubmed:publicationType | Meta-Analysis | lld:pubmed |
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