pubmed-article:21456071 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21456071 | lifeskim:mentions | umls-concept:C0376358 | lld:lifeskim |
pubmed-article:21456071 | lifeskim:mentions | umls-concept:C0023607 | lld:lifeskim |
pubmed-article:21456071 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:21456071 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
pubmed-article:21456071 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
pubmed-article:21456071 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:21456071 | pubmed:dateCreated | 2011-4-1 | lld:pubmed |
pubmed-article:21456071 | pubmed:abstractText | Recent data has shown that prostate cancer (PCA) cells are capable of producing testosterone directly from cholesterol, which may contribute to the development of castration resistance. While up-regulation of steroidogenic enzymes has been previously described during castration-resistant prostate cancer (CRPC) progression, regulation of this process is poorly defined. These data examine the role of luteinizing hormone (LH) in the regulation of steroidogenic machinery in PCA cells. | lld:pubmed |
pubmed-article:21456071 | pubmed:language | eng | lld:pubmed |
pubmed-article:21456071 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21456071 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:21456071 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21456071 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21456071 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21456071 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21456071 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21456071 | pubmed:month | Jun | lld:pubmed |
pubmed-article:21456071 | pubmed:issn | 1097-0045 | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:HawesDebraD | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:PinskiJacekJ | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:XiongShigangS | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:WangQingcaiQ | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:StanczykFrank... | lld:pubmed |
pubmed-article:21456071 | pubmed:author | pubmed-author:LiuStephen... | lld:pubmed |
pubmed-article:21456071 | pubmed:copyrightInfo | Copyright © 2010 Wiley-Liss, Inc. | lld:pubmed |
pubmed-article:21456071 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21456071 | pubmed:day | 1 | lld:pubmed |
pubmed-article:21456071 | pubmed:volume | 71 | lld:pubmed |
pubmed-article:21456071 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21456071 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21456071 | pubmed:pagination | 892-8 | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:meshHeading | pubmed-meshheading:21456071... | lld:pubmed |
pubmed-article:21456071 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21456071 | pubmed:articleTitle | Effect of luteinizing hormone on the steroidogenic pathway in prostate cancer. | lld:pubmed |
pubmed-article:21456071 | pubmed:affiliation | University of Southern California/Norris Comprehensive Cancer Center, Keck School of Medicine, Los Angeles, CA 90033, USA. pinski_j@ccnt.usc.edu | lld:pubmed |
pubmed-article:21456071 | pubmed:publicationType | Journal Article | lld:pubmed |