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pubmed-article:21083900pubmed:abstractTextDelivered systemically or natively circulating mesenchymal stem cells accumulate in injured tissues. During homing mesenchymal stem cells adhere to endothelial cells and infiltrate underlying tissue. Previously we have shown that adhesiveness of endothelial cells for mesenchymal stem cells correlates with the inhibition of mitochondrial function of endothelial cells and secretion of von Willebrand factor. We hypothesized that von Willebrand factor is an auto/paracrine regulator of endothelial cell adhesiveness and studied the effect of von Willebrand factor on adhesion of mesenchymal stem cells to endothelial cells.lld:pubmed
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pubmed-article:21083900pubmed:issn1757-6512lld:pubmed
pubmed-article:21083900pubmed:authorpubmed-author:CohenIra SISlld:pubmed
pubmed-article:21083900pubmed:authorpubmed-author:PotapovaIrina...lld:pubmed
pubmed-article:21083900pubmed:authorpubmed-author:DoroninSergey...lld:pubmed
pubmed-article:21083900pubmed:issnTypeElectroniclld:pubmed
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pubmed-article:21083900pubmed:pagination35lld:pubmed
pubmed-article:21083900pubmed:dateRevised2011-10-24lld:pubmed
pubmed-article:21083900pubmed:year2010lld:pubmed
pubmed-article:21083900pubmed:articleTitleVon willebrand factor increases endothelial cell adhesiveness for human mesenchymal stem cells by activating p38 mitogen-activated protein kinase.lld:pubmed
pubmed-article:21083900pubmed:affiliationDepartment of Physiology and Biophysics, Stony Brook University, Nicolls Road, Stony Brook, NY 11794, USA. irina.potapova@stonybrook.edu.lld:pubmed
pubmed-article:21083900pubmed:publicationTypeJournal Articlelld:pubmed