pubmed-article:20804581 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20804581 | lifeskim:mentions | umls-concept:C0002938 | lld:lifeskim |
pubmed-article:20804581 | lifeskim:mentions | umls-concept:C0017428 | lld:lifeskim |
pubmed-article:20804581 | lifeskim:mentions | umls-concept:C0013057 | lld:lifeskim |
pubmed-article:20804581 | lifeskim:mentions | umls-concept:C2348867 | lld:lifeskim |
pubmed-article:20804581 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:20804581 | pubmed:dateCreated | 2010-9-27 | lld:pubmed |
pubmed-article:20804581 | pubmed:abstractText | Diploid genomes are exquisitely balanced systems of gene expression. The dosage-compensation systems that evolved along with monosomic sex chromosomes exemplify the intricacies of compensating for differences in gene copy number by transcriptional regulation. | lld:pubmed |
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pubmed-article:20804581 | pubmed:language | eng | lld:pubmed |
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pubmed-article:20804581 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:20804581 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20804581 | pubmed:issn | 1465-6914 | lld:pubmed |
pubmed-article:20804581 | pubmed:author | pubmed-author:BeckerPeter... | lld:pubmed |
pubmed-article:20804581 | pubmed:author | pubmed-author:PrestelMatthi... | lld:pubmed |
pubmed-article:20804581 | pubmed:author | pubmed-author:FellerChristi... | lld:pubmed |
pubmed-article:20804581 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20804581 | pubmed:volume | 11 | lld:pubmed |
pubmed-article:20804581 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20804581 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20804581 | pubmed:pagination | 216 | lld:pubmed |
pubmed-article:20804581 | pubmed:dateRevised | 2011-8-29 | lld:pubmed |
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