pubmed-article:20413104 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0596030 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0521447 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C1120843 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0333516 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:20413104 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:20413104 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:20413104 | pubmed:dateCreated | 2010-8-3 | lld:pubmed |
pubmed-article:20413104 | pubmed:abstractText | Mitogen-activated protein (MAP) kinases and nuclear factor kappa B (NF-kappaB) are implicated in early stages of acute pancreatitis pathogenesis. We investigated the relationship between the p38 MAP kinase and NF-kappaB in isolated acinar cells. | lld:pubmed |
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pubmed-article:20413104 | pubmed:language | eng | lld:pubmed |
pubmed-article:20413104 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20413104 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:20413104 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20413104 | pubmed:month | Aug | lld:pubmed |
pubmed-article:20413104 | pubmed:issn | 1879-1883 | lld:pubmed |
pubmed-article:20413104 | pubmed:author | pubmed-author:CarterA... | lld:pubmed |
pubmed-article:20413104 | pubmed:author | pubmed-author:SamuelIsaacI | lld:pubmed |
pubmed-article:20413104 | pubmed:author | pubmed-author:YuanZuobiaoZ | lld:pubmed |
pubmed-article:20413104 | pubmed:author | pubmed-author:WilliardDebor... | lld:pubmed |
pubmed-article:20413104 | pubmed:author | pubmed-author:TwaitErikE | lld:pubmed |
pubmed-article:20413104 | pubmed:copyrightInfo | Published by Elsevier Inc. | lld:pubmed |
pubmed-article:20413104 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20413104 | pubmed:volume | 200 | lld:pubmed |
pubmed-article:20413104 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20413104 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20413104 | pubmed:pagination | 283-90 | lld:pubmed |
pubmed-article:20413104 | pubmed:dateRevised | 2011-10-4 | lld:pubmed |
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pubmed-article:20413104 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20413104 | pubmed:articleTitle | Nuclear factor kappa B-dependent gene transcription in cholecystokinin- and tumor necrosis factor-alpha-stimulated isolated acinar cells is regulated by p38 mitogen-activated protein kinase. | lld:pubmed |
pubmed-article:20413104 | pubmed:affiliation | Iowa City Veterans Affairs Medical Center, Iowa City, USA. | lld:pubmed |
pubmed-article:20413104 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20413104 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:20413104 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |