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| pubmed-article:20203300 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:20203300 | pubmed:dateCreated | 2010-4-15 | lld:pubmed |
| pubmed-article:20203300 | pubmed:abstractText | Enhanced endothelial permeability leading to intimal accumulation of low-density lipoproteins (LDL) stimulates the formation of atherosclerotic lesions. Histamine is known to increase vascular permeability. Whether this affects the formation of atherosclerotic lesions, however, remains elusive. | lld:pubmed |
| pubmed-article:20203300 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20203300 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20203300 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20203300 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20203300 | pubmed:month | May | lld:pubmed |
| pubmed-article:20203300 | pubmed:issn | 1524-4636 | lld:pubmed |
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| pubmed-article:20203300 | pubmed:volume | 30 | lld:pubmed |
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| pubmed-article:20203300 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20203300 | pubmed:articleTitle | Histamine H1 receptor promotes atherosclerotic lesion formation by increasing vascular permeability for low-density lipoproteins. | lld:pubmed |
| pubmed-article:20203300 | pubmed:affiliation | Cardiovascular Research, Institute of Physiology, University of Zurich, Switzerland. | lld:pubmed |
| pubmed-article:20203300 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20203300 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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