pubmed-article:20170541 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C1826623 | lld:lifeskim |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C0024623 | lld:lifeskim |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C0596263 | lld:lifeskim |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C1538598 | lld:lifeskim |
pubmed-article:20170541 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:20170541 | pubmed:dateCreated | 2010-3-22 | lld:pubmed |
pubmed-article:20170541 | pubmed:abstractText | The BMI1 oncogene is overexpressed in several human malignancies including gastric cancer. In addition to BMI1, mammalian cells also express Mel-18, which is closely related to BMI1. We have reported that Mel-18 functions as a potential tumor suppressor by repressing the expression of BMI1 and consequent downregulation of activated AKT in breast cancer cells. However, the mechanisms of BMI1 overexpression and the role of Mel-18 in other cancers are still not clear. The purpose of this study is to investigate the role of BMI1 and Mel-18 in gastric cancer. | lld:pubmed |
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pubmed-article:20170541 | pubmed:language | eng | lld:pubmed |
pubmed-article:20170541 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20170541 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20170541 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20170541 | pubmed:issn | 1476-4598 | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:LohWW | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:YaoQ YQY | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:DimriGoberdha... | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:ChengYu-FanYF | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:GuoWei-JianWJ | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:LiQianQ | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:LiuBing-YaBY | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:ZhangXiao-Wei... | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:ZhangFeng-Chu... | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:ShengYa-PingY... | lld:pubmed |
pubmed-article:20170541 | pubmed:author | pubmed-author:LuYou-WeiYW | lld:pubmed |
pubmed-article:20170541 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20170541 | pubmed:volume | 9 | lld:pubmed |
pubmed-article:20170541 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20170541 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20170541 | pubmed:pagination | 40 | lld:pubmed |
pubmed-article:20170541 | pubmed:dateRevised | 2010-9-30 | lld:pubmed |
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pubmed-article:20170541 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20170541 | pubmed:articleTitle | BMI1 and Mel-18 oppositely regulate carcinogenesis and progression of gastric cancer. | lld:pubmed |
pubmed-article:20170541 | pubmed:affiliation | Department of Medical Oncology, Cancer Hospital of Fudan University, 270 Dong An Road, Shanghai 200032, China. | lld:pubmed |
pubmed-article:20170541 | pubmed:publicationType | Journal Article | lld:pubmed |