| pubmed-article:20010862 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20010862 | lifeskim:mentions | umls-concept:C0025202 | lld:lifeskim |
| pubmed-article:20010862 | lifeskim:mentions | umls-concept:C1274040 | lld:lifeskim |
| pubmed-article:20010862 | lifeskim:mentions | umls-concept:C1521991 | lld:lifeskim |
| pubmed-article:20010862 | lifeskim:mentions | umls-concept:C0449560 | lld:lifeskim |
| pubmed-article:20010862 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:20010862 | pubmed:dateCreated | 2009-12-16 | lld:pubmed |
| pubmed-article:20010862 | pubmed:abstractText | In this issue, Hacker and colleagues provide further evidence that molecular subtypes of malignant melanoma may develop along divergent pathways. The authors did not find an association between somatic BRAF-mutant melanoma and germline melanocortin-1 receptor (MC1R) gene status. We discuss this seeming paradox in light of previous studies demonstrating strong associations. | lld:pubmed |
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| pubmed-article:20010862 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20010862 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20010862 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20010862 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20010862 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:20010862 | pubmed:issn | 1523-1747 | lld:pubmed |
| pubmed-article:20010862 | pubmed:author | pubmed-author:BerwickMarian... | lld:pubmed |
| pubmed-article:20010862 | pubmed:author | pubmed-author:ConwayKathlee... | lld:pubmed |
| pubmed-article:20010862 | pubmed:author | pubmed-author:KanetskyPeter... | lld:pubmed |
| pubmed-article:20010862 | pubmed:author | pubmed-author:BeggColin BCB | lld:pubmed |
| pubmed-article:20010862 | pubmed:author | pubmed-author:ThomasNancy... | lld:pubmed |
| pubmed-article:20010862 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20010862 | pubmed:volume | 130 | lld:pubmed |
| pubmed-article:20010862 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20010862 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20010862 | pubmed:pagination | 12-4 | lld:pubmed |
| pubmed-article:20010862 | pubmed:dateRevised | 2011-7-19 | lld:pubmed |
| pubmed-article:20010862 | pubmed:meshHeading | pubmed-meshheading:20010862... | lld:pubmed |
| pubmed-article:20010862 | pubmed:meshHeading | pubmed-meshheading:20010862... | lld:pubmed |
| pubmed-article:20010862 | pubmed:meshHeading | pubmed-meshheading:20010862... | lld:pubmed |
| pubmed-article:20010862 | pubmed:meshHeading | pubmed-meshheading:20010862... | lld:pubmed |
| pubmed-article:20010862 | pubmed:meshHeading | pubmed-meshheading:20010862... | lld:pubmed |
| pubmed-article:20010862 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20010862 | pubmed:articleTitle | Melanoma molecular subtypes: unifying and paradoxical results. | lld:pubmed |
| pubmed-article:20010862 | pubmed:affiliation | Department of Dermatology, Lineberger Comprehensive Cancer Center, University of North Carolina, Chapel Hill, North Carolina, USA. nthomas@med.unc.edu | lld:pubmed |
| pubmed-article:20010862 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20010862 | pubmed:publicationType | Comment | lld:pubmed |
| pubmed-article:20010862 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20010862 | lld:pubmed |
