pubmed-article:19850263 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19850263 | lifeskim:mentions | umls-concept:C0033085 | lld:lifeskim |
pubmed-article:19850263 | lifeskim:mentions | umls-concept:C0262950 | lld:lifeskim |
pubmed-article:19850263 | lifeskim:mentions | umls-concept:C0004561 | lld:lifeskim |
pubmed-article:19850263 | lifeskim:mentions | umls-concept:C0155626 | lld:lifeskim |
pubmed-article:19850263 | lifeskim:mentions | umls-concept:C0080309 | lld:lifeskim |
pubmed-article:19850263 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:19850263 | pubmed:dateCreated | 2009-10-23 | lld:pubmed |
pubmed-article:19850263 | pubmed:abstractText | In view of evidence that mature cells play a role in modulating the stem cell niche and thereby stem cell potential and proliferation, we hypothesized that a mature bone marrow (BM) mononuclear cell (MNC) infusion subfraction may have particular potency in promoting hematopoietic or resident stem cell-induced cardiac repair post-infarction. | lld:pubmed |
pubmed-article:19850263 | pubmed:language | eng | lld:pubmed |
pubmed-article:19850263 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19850263 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19850263 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19850263 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19850263 | pubmed:month | Oct | lld:pubmed |
pubmed-article:19850263 | pubmed:issn | 1876-7605 | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:RobinsonKeith... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:CarlessoNadia... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:PoznanskyMark... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:CuiJianhuaJ | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:ChronosNicola... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:PangWenxinW | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:WeichNadineN | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:TondatoFernan... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:UngsMarkM | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:GoodchildTrac... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:ArringtonJohn... | lld:pubmed |
pubmed-article:19850263 | pubmed:author | pubmed-author:GodwinLisaL | lld:pubmed |
pubmed-article:19850263 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19850263 | pubmed:volume | 2 | lld:pubmed |
pubmed-article:19850263 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19850263 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19850263 | pubmed:pagination | 1005-16 | lld:pubmed |
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pubmed-article:19850263 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19850263 | pubmed:articleTitle | Bone marrow-derived B cells preserve ventricular function after acute myocardial infarction. | lld:pubmed |
pubmed-article:19850263 | pubmed:affiliation | AC Therapeutics, Inc., Norcross, Georgia, USA. | lld:pubmed |
pubmed-article:19850263 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19850263 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |