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pubmed-article:19785656pubmed:abstractTextCa(2+)-activated Cl(-) currents (I(Cl(Ca))) in arterial smooth muscle cells are inhibited by phosphorylation. The Ca(2+)-activated Cl(-) channel (Cl(Ca)) blocker niflumic acid (NFA) produces a paradoxical dual effect on I(Cl(Ca)), causing stimulation or inhibition at potentials below or above 0 mV respectively. We tested whether the effects of NFA on I(Cl(Ca)) were modulated by phosphorylation.lld:pubmed
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pubmed-article:19785656pubmed:articleTitlePhosphorylation alters the pharmacology of Ca(2+)-activated Cl channels in rabbit pulmonary arterial smooth muscle cells.lld:pubmed
pubmed-article:19785656pubmed:affiliationDepartment of Pharmacology, Center of Biomedical Research Excellence (COBRE), University of Nevada School of Medicine, Reno, NV 89557-0270, USA.lld:pubmed
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