| pubmed-article:19704708 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19704708 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:19704708 | lifeskim:mentions | umls-concept:C1336789 | lld:lifeskim |
| pubmed-article:19704708 | lifeskim:mentions | umls-concept:C1332381 | lld:lifeskim |
| pubmed-article:19704708 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:19704708 | pubmed:dateCreated | 2009-8-25 | lld:pubmed |
| pubmed-article:19704708 | pubmed:abstractText | Human BRCA1 (BRreast CAncer susceptible gene1) is known to involve in cell cycle control, transcriptional regulation, DNA recombination, DNA repair and many other processes. hBARD1 (BRCA1-Associated Ring Domain 1) forms heterodimer via its N-terminal conserved RING domain with BRCA1. In Arabidopsis, two genes, At4g21070 and At1g04020, that share N-terminal RING domain and C-terminal BRCT (for BRCA1 C-Terminal) domains with no substantial similarities for other motifs, have been identified. AtBRCA1 was induced by gamma-ray while AtBARD1 was required for DNA repair. Recently, we find that AtBARD1 may function to confine WUS transcription in the shoot apical meristem organization center, together with the ATPase-dependent chromatin remodeling factor, SYD. In bard1-3 Arabidopsis knockout mutant, WUS was released to the outer layers and expressed at extremely high level comparing to wild-type. Our data suggest that BARD1 mainly function as a REPRESSOR OF WUSCHEL1 (ROW1). Extensive motif analyses carried out here showed that ROW1 possesses substantial sequence identity with a reported transcription repressor, MLL and also a potential PHD domain which recognizes histone tail codes, in its uncharacterized middle region. We suggest that ROW1 represses transcription in a chromatin-related mechanism. | lld:pubmed |
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| pubmed-article:19704708 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19704708 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19704708 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
| pubmed-article:19704708 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:19704708 | pubmed:issn | 1559-2324 | lld:pubmed |
| pubmed-article:19704708 | pubmed:author | pubmed-author:BaoL ELE | lld:pubmed |
| pubmed-article:19704708 | pubmed:author | pubmed-author:ZhuYu-XianYX | lld:pubmed |
| pubmed-article:19704708 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19704708 | pubmed:volume | 4 | lld:pubmed |
| pubmed-article:19704708 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19704708 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19704708 | pubmed:pagination | 52-4 | lld:pubmed |
| pubmed-article:19704708 | pubmed:dateRevised | 2010-1-15 | lld:pubmed |
| pubmed-article:19704708 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19704708 | pubmed:articleTitle | BARD1 may be renamed ROW1 because it functions mainly as a REPRESSOR OF WUSCHEL1. | lld:pubmed |
| pubmed-article:19704708 | pubmed:affiliation | The National Laboratory of Protein Engineering and Plant Genetic Engineering, College of Life Sciences, Peking University, Beijing, China. | lld:pubmed |
| pubmed-article:19704708 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19704708 | pubmed:publicationType | Comment | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:19704708 | lld:pubmed |
