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pubmed-article:19695307pubmed:abstractTextAccumulation of glutamate in ischaemic CNS is thought to amplify neuronal death during a stroke. Exposure of neurons to toxic glutamate concentrations causes an initial transient increase in [Ca(2+)](c) followed by a delayed increase commonly termed delayed [Ca(2+)](c) deregulation (DCD).lld:pubmed
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pubmed-article:19695307pubmed:articleTitleImpaired mitochondrial bioenergetics determines glutamate-induced delayed calcium deregulation in neurons.lld:pubmed
pubmed-article:19695307pubmed:affiliationDepartment of Molecular Neuroscience, UCL Institute of Neurology, Queen Square, London WC1N 3 BG, UK. a.abramov@ucl.ac.uklld:pubmed
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