pubmed-article:19695307 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0006675 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0027882 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0521451 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0221099 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0005486 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C0205421 | lld:lifeskim |
pubmed-article:19695307 | lifeskim:mentions | umls-concept:C1880287 | lld:lifeskim |
pubmed-article:19695307 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:19695307 | pubmed:dateCreated | 2010-2-22 | lld:pubmed |
pubmed-article:19695307 | pubmed:abstractText | Accumulation of glutamate in ischaemic CNS is thought to amplify neuronal death during a stroke. Exposure of neurons to toxic glutamate concentrations causes an initial transient increase in [Ca(2+)](c) followed by a delayed increase commonly termed delayed [Ca(2+)](c) deregulation (DCD). | lld:pubmed |
pubmed-article:19695307 | pubmed:language | eng | lld:pubmed |
pubmed-article:19695307 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19695307 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19695307 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19695307 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19695307 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19695307 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19695307 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19695307 | pubmed:month | Mar | lld:pubmed |
pubmed-article:19695307 | pubmed:issn | 0006-3002 | lld:pubmed |
pubmed-article:19695307 | pubmed:author | pubmed-author:DuchenMichael... | lld:pubmed |
pubmed-article:19695307 | pubmed:author | pubmed-author:AbramovAndrey... | lld:pubmed |
pubmed-article:19695307 | pubmed:copyrightInfo | Copyright (c) 2009 Elsevier B.V. All rights reserved. | lld:pubmed |
pubmed-article:19695307 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:19695307 | pubmed:volume | 1800 | lld:pubmed |
pubmed-article:19695307 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19695307 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19695307 | pubmed:pagination | 297-304 | lld:pubmed |
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pubmed-article:19695307 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:19695307 | pubmed:articleTitle | Impaired mitochondrial bioenergetics determines glutamate-induced delayed calcium deregulation in neurons. | lld:pubmed |
pubmed-article:19695307 | pubmed:affiliation | Department of Molecular Neuroscience, UCL Institute of Neurology, Queen Square, London WC1N 3 BG, UK. a.abramov@ucl.ac.uk | lld:pubmed |
pubmed-article:19695307 | pubmed:publicationType | Journal Article | lld:pubmed |