pubmed-article:1958592 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C1533585 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C1517806 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C0333684 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C1977882 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C0439855 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C1511695 | lld:lifeskim |
pubmed-article:1958592 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
pubmed-article:1958592 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:1958592 | pubmed:dateCreated | 1992-1-6 | lld:pubmed |
pubmed-article:1958592 | pubmed:abstractText | Chromosome in situ hybridization studies showed that the normal karyotype of leukemic cells from a patient with Ph1-negative, BCR-positive chronic myeloid leukemia (CML) concealed a complex t(9;22;20)(q34;q11;p13). The close association of 5'-BCR and 3'-ABL was demonstrated by field inversion gel electrophoresis, and in situ hybridization showed that BCR-ABL was located on the short arm of chromosome 20. Our findings further indicate that chromosome rearrangement is the cause of BCR-ABL gene fusion in leukemic cells that show a normal karyotype. Results from in situ hybridization studies were consistent with formation of the t(9;22;20) by a two step chromosomal rearrangement, but field inversion gel electrophoresis results indicated a more complex rearrangement. | lld:pubmed |
pubmed-article:1958592 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:language | eng | lld:pubmed |
pubmed-article:1958592 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:1958592 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1958592 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:1958592 | pubmed:month | Jul | lld:pubmed |
pubmed-article:1958592 | pubmed:issn | 1045-2257 | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:KennedyMM | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:Columbano-Gre... | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:MorrisCC | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:FitzgeraldPP | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:GroffenJJ | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:HeisterkampNN | lld:pubmed |
pubmed-article:1958592 | pubmed:author | pubmed-author:RomerilKK | lld:pubmed |
pubmed-article:1958592 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:1958592 | pubmed:volume | 3 | lld:pubmed |
pubmed-article:1958592 | pubmed:geneSymbol | ABL | lld:pubmed |
pubmed-article:1958592 | pubmed:geneSymbol | BCR | lld:pubmed |
pubmed-article:1958592 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:1958592 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:1958592 | pubmed:pagination | 263-71 | lld:pubmed |
pubmed-article:1958592 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:1958592 | pubmed:meshHeading | pubmed-meshheading:1958592-... | lld:pubmed |
pubmed-article:1958592 | pubmed:year | 1991 | lld:pubmed |
pubmed-article:1958592 | pubmed:articleTitle | A complex chromosome rearrangement forms the BCR-ABL fusion gene in leukemic cells with a normal karyotype. | lld:pubmed |
pubmed-article:1958592 | pubmed:affiliation | Cytogenetic and Molecular Oncology Unit, Christchurch School of Medicine, New Zealand. | lld:pubmed |
pubmed-article:1958592 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:1958592 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:1958592 | pubmed:publicationType | Case Reports | lld:pubmed |
pubmed-article:1958592 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:1958592 | lld:pubmed |