| pubmed-article:19584720 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19584720 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:19584720 | lifeskim:mentions | umls-concept:C0038435 | lld:lifeskim |
| pubmed-article:19584720 | lifeskim:mentions | umls-concept:C0028754 | lld:lifeskim |
| pubmed-article:19584720 | lifeskim:mentions | umls-concept:C0025517 | lld:lifeskim |
| pubmed-article:19584720 | lifeskim:mentions | umls-concept:C0597719 | lld:lifeskim |
| pubmed-article:19584720 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:19584720 | pubmed:dateCreated | 2009-8-18 | lld:pubmed |
| pubmed-article:19584720 | pubmed:abstractText | Chronic stress, combined with positive energy balance, may be a contributor to the increased risk for obesity, especially upper body obesity, and other metabolic diseases. This association may be mediated by alterations in the hypothalamic-pituitary-adrenal (HPA) axis. In this review, we summarize the major research that has been conducted on the role of the HPA axis in obesity and metabolic disease. | lld:pubmed |
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