| pubmed-article:19506371 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0128897 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0577631 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C0752046 | lld:lifeskim |
| pubmed-article:19506371 | lifeskim:mentions | umls-concept:C1551336 | lld:lifeskim |
| pubmed-article:19506371 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:19506371 | pubmed:dateCreated | 2009-7-15 | lld:pubmed |
| pubmed-article:19506371 | pubmed:abstractText | Monocyte chemoattractant protein 1 (MCP-1), acting in concert with its receptor chemokine receptor 2 (CCR2), promotes recruitment of macrophages into atherosclerotic plaque. We examined whether single nucleotide polymorphism (SNP) variants in the MCP-1 or CCR2 genes independently or in combination are associated with carotid artery atherosclerosis in an African American population at increased risk of vascular disease. | lld:pubmed |
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| pubmed-article:19506371 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19506371 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19506371 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19506371 | pubmed:issn | 1421-9786 | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:YanekLisa RLR | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:BeckerDiane... | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:BeckerLewis... | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:NyquistPaul... | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:McKenzieLouis... | lld:pubmed |
| pubmed-article:19506371 | pubmed:author | pubmed-author:WinklerCherie... | lld:pubmed |
| pubmed-article:19506371 | pubmed:copyrightInfo | 2009 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:19506371 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19506371 | pubmed:volume | 28 | lld:pubmed |
| pubmed-article:19506371 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19506371 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19506371 | pubmed:pagination | 124-30 | lld:pubmed |
| pubmed-article:19506371 | pubmed:dateRevised | 2010-9-27 | lld:pubmed |
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| pubmed-article:19506371 | pubmed:meshHeading | pubmed-meshheading:19506371... | lld:pubmed |
| pubmed-article:19506371 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19506371 | pubmed:articleTitle | Single nucleotide polymorphisms in monocyte chemoattractant protein-1 and its receptor act synergistically to increase the risk of carotid atherosclerosis. | lld:pubmed |
| pubmed-article:19506371 | pubmed:affiliation | Department of Anesthesiology/Critical Care Medicine, Johns Hopkins School of Medicine, Baltimore, MD 21287, USA. pnyquis1@jhmi.edu | lld:pubmed |
