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pubmed-article:19455142pubmed:abstractTextCytochrome c is a pivotal protein that resides in mitochondria as component of mitochondria respiration and apoptosis initiator. Using murine cells lacking cytochrome c, we showed here that cytochrome c-deficient cells had attenuated reactive oxygen species/nitric oxide and micronuclei induction to radiation-induced bystander signals, indicating cytochrome c is essential for the bystander effect.lld:pubmed
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pubmed-article:19455142pubmed:articleTitleMitochondrial dysfunction resulting from loss of cytochrome c impairs radiation-induced bystander effect.lld:pubmed
pubmed-article:19455142pubmed:affiliationState Key Laboratory of Nuclear Physics and Technology, Peking University, Beijing 100871, People's Republic of China.lld:pubmed
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