| pubmed-article:19438608 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C1123023 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0007570 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0011608 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0229657 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0237753 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C0175630 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C1705241 | lld:lifeskim |
| pubmed-article:19438608 | lifeskim:mentions | umls-concept:C1705242 | lld:lifeskim |
| pubmed-article:19438608 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:19438608 | pubmed:dateCreated | 2009-5-14 | lld:pubmed |
| pubmed-article:19438608 | pubmed:abstractText | The two clinical phenotypes of gluten enteropathy, coeliac disease (CD) and dermatitis herpetiformis (DH), were characterized for numbers and homing profiles of circulating final effector B cells, plasmablasts, identified as immunoglobulin (Ig)-secreting cells (ISC). In CD, the numbers of ISC were approximately 50% lower than in DH or controls. ISC expressed peripheral lymph node homing receptor (HR), L-selectin, less frequently in CD (54%) and DH (52%) patients than in controls (70%). The expression of gut mucosal HR, alpha(4)beta(7), was less frequent in CD (42%) than in DH (65%) or controls (60%). In DH, but not in CD or controls, a higher proportion of IgA1-ISC (40%) than IgA2-ISC (25%) expressed the skin HR, cutaneous lymphocyte-associated antigen. In gluten enteropathy circulating plasmablasts are more mature, but decreased in number, and have distorted homing profiles. Differential IgA1-plasmablast homing could be associated with the development of skin rash with IgA1-deposits in DH but not in CD. | lld:pubmed |
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| pubmed-article:19438608 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19438608 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19438608 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19438608 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:19438608 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19438608 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:19438608 | pubmed:issn | 1365-2249 | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:ReunalaTT | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:SavilahtiEE | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:KanteleJ MJM | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:KanteleA MAM | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:ArvilommiH... | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:Westerholm-Or... | lld:pubmed |
| pubmed-article:19438608 | pubmed:author | pubmed-author:PakkanenSS | lld:pubmed |
| pubmed-article:19438608 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19438608 | pubmed:volume | 156 | lld:pubmed |
| pubmed-article:19438608 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19438608 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19438608 | pubmed:pagination | 535-41 | lld:pubmed |
| pubmed-article:19438608 | pubmed:dateRevised | 2010-9-24 | lld:pubmed |
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