pubmed-article:19273450 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0003873 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0520817 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0065661 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:19273450 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
pubmed-article:19273450 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:19273450 | pubmed:dateCreated | 2009-4-3 | lld:pubmed |
pubmed-article:19273450 | pubmed:abstractText | To study the association between polymorphisms in the mannose-binding lectin gene (MBL2) and disease activity, physical disability, and joint erosions in patients with newly diagnosed rheumatoid arthritis (RA). | lld:pubmed |
pubmed-article:19273450 | pubmed:language | eng | lld:pubmed |
pubmed-article:19273450 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19273450 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19273450 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:19273450 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19273450 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19273450 | pubmed:month | Apr | lld:pubmed |
pubmed-article:19273450 | pubmed:issn | 0315-162X | lld:pubmed |
pubmed-article:19273450 | pubmed:author | pubmed-author:HeegaardNiels... | lld:pubmed |
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pubmed-article:19273450 | pubmed:author | pubmed-author:JacobsenSøren... | lld:pubmed |
pubmed-article:19273450 | pubmed:author | pubmed-author:SkjødtHenrikH | lld:pubmed |
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pubmed-article:19273450 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:19273450 | pubmed:volume | 36 | lld:pubmed |
pubmed-article:19273450 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19273450 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19273450 | pubmed:pagination | 731-5 | lld:pubmed |
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pubmed-article:19273450 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19273450 | pubmed:articleTitle | Mannose-binding lectin gene polymorphisms are associated with disease activity and physical disability in untreated, anti-cyclic citrullinated peptide-positive patients with early rheumatoid arthritis. | lld:pubmed |
pubmed-article:19273450 | pubmed:affiliation | Department of Rheumatology, Rigshospitalet, Copenhagen University Hospital, Blegdamsvej 9, DK-2100 Copenhagen, Denmark. sj@dadlnet.dk | lld:pubmed |
pubmed-article:19273450 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19273450 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
pubmed-article:19273450 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:19273450 | pubmed:publicationType | Multicenter Study | lld:pubmed |
entrez-gene:4153 | entrezgene:pubmed | pubmed-article:19273450 | lld:entrezgene |
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