pubmed-article:19112496 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0003374 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0205082 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C1333652 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0052432 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C1957158 | lld:lifeskim |
pubmed-article:19112496 | lifeskim:mentions | umls-concept:C0332167 | lld:lifeskim |
pubmed-article:19112496 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:19112496 | pubmed:dateCreated | 2008-12-29 | lld:pubmed |
pubmed-article:19112496 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19112496 | pubmed:abstractText | Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common inherited human enzyme defect. This deficiency provides some protection from clinical malaria, but it can also cause haemolysis after administration of drugs with oxidant properties. | lld:pubmed |
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pubmed-article:19112496 | pubmed:language | eng | lld:pubmed |
pubmed-article:19112496 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19112496 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:19112496 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19112496 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:19112496 | pubmed:author | pubmed-author:ModianoDavidD | lld:pubmed |
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pubmed-article:19112496 | pubmed:author | pubmed-author:LeeSue JSJ | lld:pubmed |
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pubmed-article:19112496 | pubmed:author | pubmed-author:AvellinoPamel... | lld:pubmed |
pubmed-article:19112496 | pubmed:author | pubmed-author:UwimanaAlineA | lld:pubmed |
pubmed-article:19112496 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19112496 | pubmed:volume | 3 | lld:pubmed |
pubmed-article:19112496 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19112496 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19112496 | pubmed:pagination | e4031 | lld:pubmed |
pubmed-article:19112496 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:19112496 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:19112496 | pubmed:articleTitle | High risk of severe anaemia after chlorproguanil-dapsone+artesunate antimalarial treatment in patients with G6PD (A-) deficiency. | lld:pubmed |
pubmed-article:19112496 | pubmed:affiliation | Centre for Vaccinology and Tropical Medicine, Nuffield Department of Medicine, Oxford University, Oxford, United Kingdom. Caterina.Fanello@ndm.ox.ac.uk | lld:pubmed |
pubmed-article:19112496 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19112496 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
pubmed-article:19112496 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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