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pubmed-article:19101695pubmed:abstractTextIntimal hyperplasia remains the principal lesion in the development of restenosis after vessel wall injury. Modulation of the extracellular matrix by proteases is a pivotal component of the response to injury. The aim of this study is to characterize the changes in gelatinase (MMP-2/TIMP-2 and MMP-9/TIMP-1) systems in a murine model.lld:pubmed
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pubmed-article:19101695pubmed:articleTitlePatterns of gelatinase activation induced by injury in the murine femoral artery.lld:pubmed
pubmed-article:19101695pubmed:affiliationVascular Biology and Therapeutics Program, Methodist DeBakey Heart and Vascular Center, Department of Cardiovascular Surgery, The Methodist Hospital, Houston, Texas 77030, USA.lld:pubmed
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