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| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C0027021 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C0006560 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C0030685 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C0680255 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C0391871 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C1283071 | lld:lifeskim |
| pubmed-article:19074520 | lifeskim:mentions | umls-concept:C1963578 | lld:lifeskim |
| pubmed-article:19074520 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:19074520 | pubmed:dateCreated | 2009-1-30 | lld:pubmed |
| pubmed-article:19074520 | pubmed:abstractText | C-reactive protein (CRP), the prototypic marker of inflammation, is present in atherosclerotic plaques and appears to promote atherogenesis. Also, CRP has been localized to monocytes and tissue macrophages, which are present in the necrotic core of lesions prone to plaque rupture. Leukocyte-derived myeloperoxidase (MPO), primarily hosted in human polymorphonuclear cells (PMNs), has also been shown to be present in human atherosclerotic lesions. Because MPO and CRP concentrations are increased in acute coronary syndrome (ACS) patients and predict poor outcomes, we tested the effect of CRP on MPO release from PMNs and monocytes. | lld:pubmed |
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| pubmed-article:19074520 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19074520 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19074520 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19074520 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:19074520 | pubmed:issn | 1530-8561 | lld:pubmed |
| pubmed-article:19074520 | pubmed:author | pubmed-author:JialalIshwarl... | lld:pubmed |
| pubmed-article:19074520 | pubmed:author | pubmed-author:DevarajSridev... | lld:pubmed |
| pubmed-article:19074520 | pubmed:author | pubmed-author:UmaSinghS | lld:pubmed |
| pubmed-article:19074520 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19074520 | pubmed:volume | 55 | lld:pubmed |
| pubmed-article:19074520 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19074520 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19074520 | pubmed:pagination | 361-4 | lld:pubmed |
| pubmed-article:19074520 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:19074520 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19074520 | pubmed:articleTitle | C-reactive protein stimulates myeloperoxidase release from polymorphonuclear cells and monocytes: implications for acute coronary syndromes. | lld:pubmed |
| pubmed-article:19074520 | pubmed:affiliation | Department of Medical Pathology and Laboratory Medicine, UC Davis Medical Center, Sacramento, CA 95817, USA. uma.singh@ucdmc.ucdavis.edu | lld:pubmed |
| pubmed-article:19074520 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19074520 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:19074520 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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