| pubmed-article:19013555 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0004927 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0170657 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0301630 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C1555903 | lld:lifeskim |
| pubmed-article:19013555 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
| pubmed-article:19013555 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:19013555 | pubmed:dateCreated | 2009-2-9 | lld:pubmed |
| pubmed-article:19013555 | pubmed:abstractText | Obsessive-compulsive disorder (OCD) is characterized by intrusive thoughts, images, or impulses and/or repetitive stereotypical behavior. Obsessive-compulsive disorder patients exhibit reduced prepulse inhibition (PPI) and symptom exacerbation after challenge with 5-HT1B receptor agonists. Recently, gain-of-function alleles of the serotonin transporter (5-HTT) have been associated with OCD. We tested the hypothesis that reducing 5-HTT function chronically, either genetically or via serotonin reuptake inhibitor (SRI) treatment, attenuates PPI deficits and perseverative hyperlocomotion induced by 5-HT1B agonists in mice. | lld:pubmed |
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| pubmed-article:19013555 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19013555 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19013555 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19013555 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:19013555 | pubmed:issn | 1873-2402 | lld:pubmed |
| pubmed-article:19013555 | pubmed:author | pubmed-author:WaeberChristi... | lld:pubmed |
