| pubmed-article:18985293 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18985293 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:18985293 | lifeskim:mentions | umls-concept:C0033975 | lld:lifeskim |
| pubmed-article:18985293 | lifeskim:mentions | umls-concept:C0036341 | lld:lifeskim |
| pubmed-article:18985293 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:18985293 | lifeskim:mentions | umls-concept:C0871125 | lld:lifeskim |
| pubmed-article:18985293 | pubmed:dateCreated | 2008-11-5 | lld:pubmed |
| pubmed-article:18985293 | pubmed:abstractText | Decomposition of schizophrenia into neurobiological vulnerability traits is necessary to understand the complex genetic underpinnings of this phenomenologically defined disorder. This issue is discussed with a focus on prepulse inhibition (PPI) as a neurobiological phenotype and the 5HT2a-receptor as a candidate gene. A series of recent studies illuminates that PPI and 5HT2a-receptors present as vulnerability markers for schizophrenia; a functional sequence variant in the 5HT2a-gene is contributing to this relationship and might consequently contribute to the genetic predisposition to schizophrenia with a very small risk increase. | lld:pubmed |
| pubmed-article:18985293 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18985293 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18985293 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:18985293 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18985293 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18985293 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18985293 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:18985293 | pubmed:issn | 0940-1334 | lld:pubmed |
| pubmed-article:18985293 | pubmed:author | pubmed-author:MaierWolfgang... | lld:pubmed |
| pubmed-article:18985293 | pubmed:author | pubmed-author:WagnerMichael... | lld:pubmed |
| pubmed-article:18985293 | pubmed:author | pubmed-author:MössnerRainal... | lld:pubmed |
| pubmed-article:18985293 | pubmed:author | pubmed-author:HurlemannRené... | lld:pubmed |
| pubmed-article:18985293 | pubmed:author | pubmed-author:QuednowBoris... | lld:pubmed |
| pubmed-article:18985293 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18985293 | pubmed:volume | 258 Suppl 5 | lld:pubmed |
| pubmed-article:18985293 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18985293 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18985293 | pubmed:pagination | 40-3 | lld:pubmed |
| pubmed-article:18985293 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:meshHeading | pubmed-meshheading:18985293... | lld:pubmed |
| pubmed-article:18985293 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18985293 | pubmed:articleTitle | From genes to psychoses and back: the role of the 5HT2alpha-receptor and prepulse inhibition in schizophrenia. | lld:pubmed |
| pubmed-article:18985293 | pubmed:affiliation | Klinik und Poliklinik für Psychiatrie und Psychotherapie, der Universität Bonn, Sigmund-Freud-Strasse 25, Bonn, Germany. Wolfgang.Maier@ukb.uni-bonn.de | lld:pubmed |
| pubmed-article:18985293 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18985293 | pubmed:publicationType | Review | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:18985293 | lld:pubmed |
