| pubmed-article:18816180 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0004623 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0027950 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0021965 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C1171362 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0332448 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0205178 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C1515670 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:18816180 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:18816180 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:18816180 | pubmed:dateCreated | 2008-10-10 | lld:pubmed |
| pubmed-article:18816180 | pubmed:abstractText | Macrophages and polymorphonuclear cells (PMNs) play an important role in the first line of defense against bacteria by infiltrating the infected organ in order to clear the harmful pathogen. Our earlier studies showed that granulocytes express type 3 deiodinase (D3) when activated during a turpentine-induced abscess. We hypothesized that D3 expression by granulocytes may also occur during bacterial infection. | lld:pubmed |
| pubmed-article:18816180 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18816180 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18816180 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:18816180 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18816180 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18816180 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18816180 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:18816180 | pubmed:issn | 1557-9077 | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:WielandCathar... | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:BoelenAnitaA | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:KwakkelJoanJ | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:VisserTheo... | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:FliersEricE | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:WiersingaWilm... | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:RenckensRosem... | lld:pubmed |
| pubmed-article:18816180 | pubmed:author | pubmed-author:BoorsmaJeffre... | lld:pubmed |
| pubmed-article:18816180 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18816180 | pubmed:volume | 18 | lld:pubmed |
| pubmed-article:18816180 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18816180 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18816180 | pubmed:pagination | 1095-103 | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:meshHeading | pubmed-meshheading:18816180... | lld:pubmed |
| pubmed-article:18816180 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18816180 | pubmed:articleTitle | Type 3 deiodinase is highly expressed in infiltrating neutrophilic granulocytes in response to acute bacterial infection. | lld:pubmed |
| pubmed-article:18816180 | pubmed:affiliation | Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands. a.boelen@amc.uva.nl | lld:pubmed |
| pubmed-article:18816180 | pubmed:publicationType | Journal Article | lld:pubmed |
| entrez-gene:107585 | entrezgene:pubmed | pubmed-article:18816180 | lld:entrezgene |
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