| pubmed-article:18651010 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0043393 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0020205 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0009015 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0077401 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0597298 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C1523493 | lld:lifeskim |
| pubmed-article:18651010 | lifeskim:mentions | umls-concept:C0449719 | lld:lifeskim |
| pubmed-article:18651010 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:18651010 | pubmed:dateCreated | 2008-7-24 | lld:pubmed |
| pubmed-article:18651010 | pubmed:abstractText | To elucidate the molecular pathogenesis behind increased levels of laminin in cardiac muscle cells in cardiomyopathy by using a yeast hybrid screen. The present study reports the cloning of a newly identified heart-specific troponin I isoform, which is putatively linked to laminin. Future studies will explore the functional significance of this connection. | lld:pubmed |
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| pubmed-article:18651010 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18651010 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18651010 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
| pubmed-article:18651010 | pubmed:issn | 1205-6626 | lld:pubmed |
| pubmed-article:18651010 | pubmed:author | pubmed-author:ItoMasakiM | lld:pubmed |
| pubmed-article:18651010 | pubmed:author | pubmed-author:SuzukiHideaki... | lld:pubmed |
| pubmed-article:18651010 | pubmed:author | pubmed-author:YamadaHisashi... | lld:pubmed |
| pubmed-article:18651010 | pubmed:author | pubmed-author:Horiguchi-Yam... | lld:pubmed |
| pubmed-article:18651010 | pubmed:author | pubmed-author:ArakawaYasuhi... | lld:pubmed |
| pubmed-article:18651010 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18651010 | pubmed:volume | 11 | lld:pubmed |
| pubmed-article:18651010 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18651010 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18651010 | pubmed:pagination | 4-7 | lld:pubmed |
| pubmed-article:18651010 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:18651010 | pubmed:articleTitle | Cloning of a newly identified heart-specific troponin I isoform, which lacks the troponin T binding portion, using the yeast hybrid system. | lld:pubmed |
| pubmed-article:18651010 | pubmed:affiliation | Departments of Internal Medicine and. | lld:pubmed |
| pubmed-article:18651010 | pubmed:publicationType | Journal Article | lld:pubmed |
