pubmed-article:18645055 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18645055 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:18645055 | lifeskim:mentions | umls-concept:C0870134 | lld:lifeskim |
pubmed-article:18645055 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
pubmed-article:18645055 | lifeskim:mentions | umls-concept:C0225828 | lld:lifeskim |
pubmed-article:18645055 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:18645055 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:18645055 | pubmed:dateCreated | 2008-8-5 | lld:pubmed |
pubmed-article:18645055 | pubmed:abstractText | Increasing evidence, derived mainly from animal models, supports the existence of endogenous cardiac renewal and repair mechanisms in adult mammalian hearts that could contribute to normal homeostasis and the responses to pathological insults. | lld:pubmed |
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pubmed-article:18645055 | pubmed:language | eng | lld:pubmed |
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pubmed-article:18645055 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:18645055 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18645055 | pubmed:month | Aug | lld:pubmed |
pubmed-article:18645055 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:JaleelNaserN | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:HouserSteven... | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:WangHongmeiH | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:KuboHajimeH | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:MarguliesKenn... | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:BerrettaRemus... | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:ShanXiaoyinX | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:BratinovGeorg... | lld:pubmed |
pubmed-article:18645055 | pubmed:author | pubmed-author:KumarapeliAsa... | lld:pubmed |
pubmed-article:18645055 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18645055 | pubmed:day | 5 | lld:pubmed |
pubmed-article:18645055 | pubmed:volume | 118 | lld:pubmed |
pubmed-article:18645055 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18645055 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18645055 | pubmed:pagination | 649-57 | lld:pubmed |
pubmed-article:18645055 | pubmed:dateRevised | 2011-6-9 | lld:pubmed |
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pubmed-article:18645055 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18645055 | pubmed:articleTitle | Increased cardiac myocyte progenitors in failing human hearts. | lld:pubmed |
pubmed-article:18645055 | pubmed:affiliation | Department of Physiology, Temple University School of Medicine, Philadelphia, PA, USA. | lld:pubmed |
pubmed-article:18645055 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18645055 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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