| pubmed-article:18598622 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18598622 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:18598622 | lifeskim:mentions | umls-concept:C0003062 | lld:lifeskim |
| pubmed-article:18598622 | lifeskim:mentions | umls-concept:C0016452 | lld:lifeskim |
| pubmed-article:18598622 | lifeskim:mentions | umls-concept:C0079134 | lld:lifeskim |
| pubmed-article:18598622 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:18598622 | pubmed:dateCreated | 2008-7-7 | lld:pubmed |
| pubmed-article:18598622 | pubmed:abstractText | Clostridium difficile is a recognized pathogen in neonatal pigs and may contribute to enteritis in calves. Toxinotype V strains have been rare causes of human C. difficile-associated disease (CDAD). We examined toxinotype V in human disease, the genetic relationship of animal and human toxinotype V strains, and in vitro toxin production of these strains. From 2001 through 2006, 8 (1.3%) of 620 patient isolates were identified as toxinotype V; before 2001, 7 (<0.02%) of approximately 6,000 isolates were identified as toxinotype V. Six (46.2%) of 13 case-patients for whom information was available had community-associated CDAD. Molecular characterization showed a high degree of similarity between human and animal toxinotype V isolates; all contained a 39-bp tcdC deletion and most produced binary toxin. Further study is needed to understand the epidemiology of CDAD caused by toxinotype V C. difficile, including the potential of foodborne transmission to humans. | lld:pubmed |
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| pubmed-article:18598622 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18598622 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18598622 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18598622 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18598622 | pubmed:issn | 1080-6059 | lld:pubmed |
| pubmed-article:18598622 | pubmed:author | pubmed-author:McDonaldL... | lld:pubmed |
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| pubmed-article:18598622 | pubmed:author | pubmed-author:WarnyMichaelM | lld:pubmed |
| pubmed-article:18598622 | pubmed:author | pubmed-author:LimbagoBrandi... | lld:pubmed |
| pubmed-article:18598622 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18598622 | pubmed:volume | 14 | lld:pubmed |
| pubmed-article:18598622 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18598622 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18598622 | pubmed:pagination | 1039-45 | lld:pubmed |
| pubmed-article:18598622 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:18598622 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18598622 | pubmed:articleTitle | Toxinotype V Clostridium difficile in humans and food animals. | lld:pubmed |
| pubmed-article:18598622 | pubmed:affiliation | Centers for Disease Control and Prevention, Atlanta, Georgia, USA. | lld:pubmed |
| pubmed-article:18598622 | pubmed:publicationType | Journal Article | lld:pubmed |
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